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Curr Treat Options Gastroenterol. 1998 Dec;1(1):20-26.
Idiopathic Constipation and Fecal Incontinence.

Krevsky B.

Gastroenterology Section, Temple University Hospital, 3401 North Broad Street, Philadelphia, PA 19140.

Patients with constipation or fecal incontinence have great potential for remediation. Both disorders warrant a thorough diagnostic evaluation to search for remediable causes, especially to identify defecation disorders. Constipation should be treated by relieving any impactions and then starting a course of fiber supplementation of at least 20 g per day. If not successful, other agents such as lactulose, polyethylene glycol solutions, or stimulants can be added. Biofeedback may be very helpful. If all else fails, several surgical procedures are available. Fecal incontinence should be treated by fiber supplements, encouragement of regularity, and enemas as needed. Kegel exercises and biofeedback are often helpful, but surgery may be needed and is most effective in recent onset or traumatically induced incontinence.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=11096560&dopt=Abstract [PubMed - as supplied by publisher]

Curr Treat Options Gastroenterol. 1999 Feb;2(1):13-19.
Irritable Bowel Syndrome.

Wald A.

University of Pittsburgh Medical Center, Pittsburgh University Hospital, Mezzanine Level, C-Wing, 200 Lothrop Street, Pittsburgh, PA 15213-2582.

I believe there are four essential elements in the management of patients with irritable bowel syndrome (IBS): to establish a good physician-patient relationship; to educate patients about their condition; to emphasize the excellent prognosis and benign nature of the illness; and to employ therapeutic interventions centering on dietary modifications, pharmacotherapy, and behavioral strategies tailored to the individual. Initially, I establish the diagnosis, exclude organic causes, educate patients about the disease, establish realistic expectations and consistent limits, and involve patients in disease management. I find it critical to determine why the patient is seeking assistance (eg, cancer phobia, disability, interpersonal distress, or exacerbation of symptoms). Most patients can be treated by their primary care physician. However, specialty consultations may be needed to reinforce management strategies, perform additional diagnostic tests, or institute specialized treatment. Psychological co-morbidities do not cause symptoms but do affect how patients respond to them and influence health care-seeking behavior. I find that these issues are best explored over a series of visits when the physician-patient relationship has been established. It can be helpful to have patients fill out a self-administered test to identify psychological co-morbidities. I often use these tests as a basis for extended inquiries into this area, resulting in the initiation of appropriate therapies. I encourage patients to keep a 2-week diary of food intake and gastrointestinal symptoms. In this way, patients become actively involved in management of their disease, and I may be able to obtain information from the diary that will be valuable in making treatment decisions. I do not believe that diagnostic studies for food intolerances are cost-effective or particularly helpful; however, exclusion diets may be beneficial. I introduce fiber supplements gradually and monitor them for tolerance and palatability. Synthetic fiber is often better-tolerated than natural fiber, but must be individualized. In my experience, excessive fiber supplementation often is counterproductive, as abdominal cramps and bloating may worsen. Antidiarrheal agents are very effective when used correctly, preferably in divided doses. I use them in patients in anticipation of diarrhea and especially in those who fear symptoms when engaged in activities outside the home. I encourage patients to make decisions as to when and how much to use. However, almost always, a morning dose before breakfast is used (loperamide, 2 to 6 mg) and, perhaps again later in the day when symptoms of diarrhea are prominent. I prefer antispasmodics to be used intermittently in response to periods of increased abdominal pain, cramps, and urgency. For patients with daily symptoms, especially after meals, agents such as dicyclomine before meals are useful. For patients with infrequent but severe episodes of unpredictable pain, sublingual hyoscyamine often produces rapid relief and instills confidence. In general, I recommend that oral antispasmodics be used for a limited period of time rather than indefinitely, and generally for periods of time when symptoms are prominent. For chronic visceral pain syndromes, I recommend small doses of tricyclic antidepressants. These agents are especially effective in diarrhea-predominant patients with disturbed sleep patterns but may be unacceptable to patients with constipation. I educate patients that side effects occur early and benefits may not be apparent for 3 to 4 weeks. I consider using SSRIs in low doses in patients with constipation-predominant IBS; cisapride, 10 to 20 mg three times per day, also may be beneficial. When taken with drugs that inhibit cytochrome P450, cisapride has been associated with serious cardiac arrhythmias caused by QT prolongation, including ventricular arrhythmias and torsades de pointes. These drugs include the azole fungicides; erythromycin, clarithromycin, and troleandomycin; some antidepressants; HIV protease inhibitors; and others. In patients with IBS with mild to moderate co-morbid depression, I have found that the use of SSRIs such as paroxetine, fluoxetine, or sertraline may be beneficial. It is important to tell patients that anxiety and disturbed sleep may occur during the first 10 days and benefits may not occur for 3 to 4 weeks. I prescribe a small amount of a short-acting benzodiazepine such as alprazolam, 0.5 mg two times per day, to control these symptoms. For generalized anxiety without depression, buspirone or clonazepam may be useful. I have found that patients who also have associated panic disorder may benefit from a benzodiazepine, tricyclic antidepressant, or an SSRI. However, these patients are best managed in conjunction with a psychiatrist or psychologist. I consider the use of alternative therapies in patients who fail to respond to conventional measures and who are receptive to alternative strategies. These include general relaxation techniques such as biofeedback and hypnosis therapies.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=11096567&dopt=Abstract [PubMed - as supplied by publisher]

Curr Treat Options Neurol. 2000 Jan;2(1):23-30.
Diabetic Neuropathies.

Zochodne DW.

Department of Clinical Neurosciences, University of Calgary, Room 182A, 3330 Hospital Drive, N.W., Calgary, Alberta T2N 4N1, Canada.

There are currently no treatments available (beyond optimal control of hyperglycemia) that arrest or reverse progressive diabetic polyneuropathy. Consultation with a diabetologist is indicated for patients with poorly controlled disease and polyneuropathy. Immunotherapy for diabetic lumbosacral plexopathy has been advocated but is not supported to date by class 1 clinical trial evidence. Pharmacologic treatment for painful neuropathy may include topical anesthetics, capsaicin cream, anticonvulsants, tricyclic antidepressants, mexiletine, and opioids. Gabapentin, a newer anticonvulsant, has an attractive side-effect profile (a consideration in older patients) and has fewer interactions with other drugs. Amitriptyline may cause excessive sedation, postural hypotension, constipation, and urinary retention, but low evening doses (10 to 25 mg), slowly titrated upward, may offer relief from nocturnal pain. Opioids should not be withheld from patients with severe pain or with intolerance of or contraindications to other agents. A single physician should supervise their use.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=11096734&dopt=Abstract [PubMed - as supplied by publisher]

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