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Alcohol Clin Exp Res. 2002 May;26(5):617-26.
Alcohol consumption promotes body weight loss in melanoma-bearing mice.

Nunnez NP, Carter PA, Meadows GG.

Department of Pharmaceutical Sciences, the Pharmacology and Toxicology Graduate Program, the Cancer Prevention & Research Center, Washington State University, Pullman, Washington 99164-6510, USA.

BACKGROUND: Alcohol consumption is an important risk factor for cancer. Little is known about its effects on cancer progression. Previously, we showed that high ethanol consumption inhibited metastasis of B16BL6 melanoma-bearing mice without affecting primary tumor growth. On the other hand, ethanol-consuming tumor-bearing (TE) mice exhibited decreased survival and decreased body weight as compared to water-drinking, tumor-bearing (TW) mice. The focus of this study was to determine how alcohol promotes weight loss in melanoma-bearing mice. METHODS: Female, C57BL/6 mice were given water or 20% w/v ethanol in the drinking water for 3 weeks to 6 months before subcutaneous inoculation of 1 x 10(6) B16BL6 melanoma cells. Mice continued to receive the same fluids. Biochemical parameters were evaluated at various times after tumor inoculation. Body weight, water content, tumor weight and carcass fat content were determined at necropsy. RESULTS: TW mice elicted a modest weight loss. This response was magnified 2-fold by alcohol consumption. The weight loss in TE mice is not caused by dehydration, decreased energy intake, or loss of skeletal muscle mass. It resulted specifically from loss in body fat. Other alterations associated with the fat loss in TE mice were: (1) decreased glucose, (2) elevated fatty acids, (3) elevated beta-hydroxybutyrate, (4) elevated glucagon, and (5) increased leptin levels in plasma. Body temperature decreased about 2.9 degrees C in TE mice. Metabolic rate increased in TW mice. The fat loss due to alcohol consumption in tumor-bearing mice was not due to increased metabolic rate. CONCLUSIONS: The response elicited by alcohol consumption in tumor-bearing mice is complex and associated with alterations in metabolism and hormones. These findings suggest that alcohol abuse could be a risk factor for cancer patients because it invokes a strong depletion of body fat. This could facilitate wasting and shorten survival time.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12045469&dopt=Abstract

Naturwissenschaften. 2002 Feb;89(2):60-6.
A chemical view of the most ancient metazoa--biomarker chemotaxonomy of hexactinellid sponges.

Thiel V, Blumenberg M, Hefter J, Pape T, Pomponi S, Reed J, Reitner J, Worheide G, Michaelis W.

Institut fur Biogeochemie und Meereschemie, Universitat Hamburg, Germany. thieeowiss.uni-hamburg.de

Hexactinellid sponges are often considered to be the most ancient metazoans. Lipid biomarkers from 23 species were studied for information on their phylogenetic properties, particularly their disputed relation to the two other sponge classes (Demospongiae, Calcarea). The most prominent lipid compounds in the Hexactinellida comprise C28 to C32 polyenoic fatty acids. Their structures parallel the unique patterns found in demosponge membrane fatty acids ('demospongic acids') and strongly support a close phylogenetic association of the Demospongiae and the Hexactinellida. Both taxa also show unusual mid-chain methylated fatty acids (C15-C25) and irregular C25- and C40-isoprenoid hydrocarbons, tracers for specific eubacteria and Archaea, respectively. These biomarkers indicate a similar, highly conservative symbiont community, although some shift in the abundance of the associated microbiota was observed. The lack of these features in calcareous sponges further contradicts the still common view that Calcarea and Demospongiae are more closely related to each other than either is to the Hexactinellida.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12046622&dopt=Abstract

Eur J Clin Invest. 2002 Sep;32(9):707-12.
Molecular study of electron transfer flavoprotein alpha-subunit deficiency in two Japanese children with different phenotypes of glutaric acidemia type II.

Purevjav E, Kimura M, Takusa Y, Ohura T, Tsuchiya M, Hara N, Fukao T, Yamaguchi S.

Department of Pediatric, Shimane Medical University, Izumo, Shimane, Japan.

BACKGROUND: Electron transfer flavoprotein is a mitochondrial matrix protein composed of alpha- and beta-subunits (ETF alpha and ETF beta, respectively). This protein transfers electrons between several mitochondrial dehydrogenases and the main respiratory chain via ETF dehydrogenase (ETF-DH). Defects in ETF or ETF-DH cause glutaric acidemias type II (GAII). MATERIALS AND METHODS: We investigated the molecular basis of ETF alpha deficiency in two Japanese children with different clinical phenotypes using expression study. RESULTS: Patient 1 had the severe form of GAII, a compound heterozygote of two mutations: 799G to A (alpha G267R) and nonsense 7C to T (alpha R3X). Patient 2 had the mild form and carried two heterozygous mutations: 764G to T (alpha G255V) and 478delG (frameshift). Both patients had one each of missense mutations in one allele; the others were either nonsense or truncated. Restriction enzyme digestion assay using genomic DNAs from 100 healthy Japanese revealed that these mutations were all novel. No signal for ETF alpha was detected by immunoblotting in cases of missense mutants, while wild-type cDNA resulted in expression of ETF alpha protein. Transfection with wild-type ETF alpha cDNA into cultured cells from both patients elevated incorporation of radioisotope-labelled fatty acids. CONCLUSION: These four mutations were pathogenic for GAII and missense mutations, alpha G255V and alpha G267R were considered anecdotal for mild and severe forms, respectively.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12486872&dopt=Abstract

J Environ Sci Health Part A Tox Hazard Subst Environ Eng. 2002;37(4):439-49.
Volatile fatty acids as electron donors for the reductive dechlorination of chloroethenes.

Lu XX, Li GH, Tao S, Bosma T, Gerritse J.

Tsinghua University, Beijing, PR China. Luxail.tsinghua.edu.cn

Uses of a mixture of six volatile fatty acids (VFAs) including acetic, propionic, butyric, isobutyric, valeric and isovaleric acids as electron donors for the reductive dechlorination of chloroethenes have been investigated by both microcosm and column studies. The fates of tetrachloroethene (PCE), cis-dichloroethene (cis-DCE) and vinyl chloride (VC) in the presence of VFAs and in the absence of VFAs were respectively documented. The results showed that VFAs stimulated complete reductive dechlorination of chloroethenes, either as direct substrates for the dechlorinating bacteria or via H2 formed during VFAs-degradation. There were sequential utilizations of different VFAs by fermenting bacteria. In the microcosm, propionic acid was the first to be used, followed by acetic, butyric, isobutyric, valeric, and isovaleric acids, and their mean first-order degradation rates obtained were 0.128, 0.048, 0.016, 0.027, 0.025 and 0.003 day(-1), respectively. In the column, propionic acid was the first to be used, followed by butyric and valeric acids, and their calculated first-order degradation rates were 0.833, 0.403 and 0.260 day(-1), respectively.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12046645&dopt=Abstract

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