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Drug Ther (NY). 1977 October;2(10):61-3.
Drug interference with laboratory tests: oral contraceptives.

Sher PP.

PIP: Changes in serum protein levels are produced by oral contraceptives. They reflect hepatic synthesis of proteins. Changes range from a 40% decrease in orosomucoid to an 180% increase in ceruloplasmin. Alterations in the concentration of some globulin proteins lead to altered serum levels of some hormones and trace metals. Plasma levels of cortisol and thyroxine are most affected. An estrogen-induced increase in thyroxine-binding globulin causes a transient reduction in free thyroxine (T4). This results in a compensatory increase in thyroid-stimulating hormone. The net effect is an unchanged concentration of T4 and the patient may be reported as euthyr id. Increases in renin substrate with rise in renin activity, angiotensin 2, and aldosterone, may be related to the development of hypertension in some women. Oral contraceptives may cause significant increases in some lipids. Elevations in cholesterol and nonesterified fatty acids are less changed. The drug-induced changes can complicate a definite diagnosis of hyperlipidemia. The glucose tolerance curve in users of oral contraceptives may simulate the diabetic type. Hepatic function tests may suggest hepatic damage. There is a higher incidence of cholecystitis and gallstones in women using oral contraceptives. Blood coagulation tests may be modified. Drug interference from oral contraceptive use must be considered in interpreting laboratory reports.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12260155&dopt=Abstract

Child Trop. 1982;(138-140):1-78.
How to feed young children.

Masse-raimbault AM.

PIP: Changes have occurred in the ways of feeding young children in the past 20 years, both in the industrialized countries and in the 3rd world. The question arises as to whether these changes are always beneficial, particularly in the developing countries. In this discussion of how to feed young children a short reminder is provided of the recommended nutritional intakes during the 1st years of life. Subsequent discussion focuses on the following: the building, energy giving, and protective foods; slight, moderate, and severe forms of malnutrition; breastfeeding (the physiology of breastfeeding, arguments favoring breastfeeding; breastfeeding and reproduction; breastfeeding successfully, and breastfeeding and medication); and artificial feeding or substitute feeding. Nutritional intakes must cover what is spent to maintain the body under various circumstances. Children have considerable, specific food requirements. These change rapidly from week to week as the young organism adjusts to an air filled world and copes with its weight and height growth and overall development. The fantastic growth that occurs during the 1st years of life explains why the young child is so vulnerable. The child must have a nutritional intake which is both quantitatively and qualitatively appropriate to its needs. For several months the child should be given a rather monotonous liquid diet. The average energy intake for the 1st year of life should be about 110 Kcal/kg/24 hours. Protein requirements are particularly high during the 1st years of life, for children must not only maintain, repair, and replace tissue, but also provide for the growth and development of their body, i.e., the building of body tissue. An important essential fatty acid is linoleic acid, which serves as a basis for the production o other essential fatty acids. Recommended intakes for calcium, iron, and vitamin A are reviewed. Analysis of food consumption shows that some populations in developing countries have a diet based essentially on 1 staple food. Most are energy giving foods and almost all are carbohydrates. The types of malnutrition caused by deficiency and the most frequently encountered in the developing countries are protein calorie malnutrition, avitaminoses, and mineral and iron deficiencies. Toward the age of 4-6 months, the mother's milk is no longer sufficient, quantitatively or qualitatively, for the satisfaction of the child's nutritional requirements and cannot ensure his/her adequate growth. In general, at this time of life the child should gradually begin to receive complemental food.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12265411&dopt=Abstract

Philipp J Nutr. 1982 Jul-Sep;35(3):106-11.
Maternal nutrition, infant health, and subsequent fertility.

Osteria TS.

PIP: This paper focuses on the influence of maternal nutrition on infant survival and growth and on subsequent fertility. During pregnancy, modifications in the maternal hormonal system help maintain availability of nutrients to the fetus relatively independently of maternal nutrition. When maternal dietary deficiencies reach critical levels, the effectiveness of these mechanisms in maintaining fetal nutrition decreases. Studies have confirmed the correlation of severe maternal malnutrition and lowered birth weight, neurological disorders, impaired physical growth, mental retardation, and poor school performance. Marginal nutritional status affects milk production capacity. The basis of most recommendations for lactating mothers is that the average mother produces 850 ml of milk each day. Depending on the anticipatory reserves of fat from pregnancy still available and the level of physical activity of the mother, from 2750 to 3300 Kcal/day may be needed. When the mother's energy intake is good, milk fat resembles that of her dietary fat, but when there is a shortage of food energy, the milk fatty acid pattern resembles the mother's subcutaneous fat stores. The lactose content and overall protein content of milk seem stable despite changes in the maternal diet, but the vitamin content, particularly water soluble vitamins, is very sensitive to dietary intake. Birth weight has consistently been found to be associated with infant mortality through 2 main mechanisms: maternal malnutrition may lead to a smaller placental size and decreased nutrient supply to the fetus, resulting in developmental retardation during intrauterine life, or maternal malnutrition may result in suboptimal lactation performance which will contribute to malnutrition and growth retardation of the child. Maternal nutritional status has an independent effect on the duration of postpartum amenorrhea. Improving maternal diet without concurrently introducing contraception may shorten the birth interval and consequently elevate the birthrate.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12266149&dopt=Abstract

Syst Appl Microbiol. 1999 Feb;22(1):113-8.
Importance of Xanthobacter autotrophicus in toluene biodegradation within a contaminated stream.

Tay ST, Hemond HF, Polz MF, Cavanaugh CM, Krumholz LR.

Ralph M Parsons Laboratory, Department of Civil and Environmental Engineering, Massachusetts Institute of Technology, Cambridge, USA.

Toluene-degrading strains T101 and T102 were isolated from rock surface biomass in a toluene-contaminated freshwater stream. These organisms were present at a density of 5.5 x 10(6) cells/g of rock surface biomass. Both are aerobic, rod-shaped, Gram-negative, non-motile, catalase-positive, oxidase-positive, with yellow pigments, and can grow on benzene. Phylogenetic analyses show that strains T101 and T102 have 16S rDNA sequences identical to Xanthobacter autotrophicus. Fatty acid analyses indicate that they are different strains of the same species Xanthobacter autotrophicus, and that they have high levels of cis-11-octadecenoic acid and cis-9-hexadecenoic acid; 3-hydroxyhexadecanoic acid is the major hydroxy fatty acid present. Strains T101 and T102 had maximal velocities (Vmax) for toluene biodegradation of 3.8 +/- 0.5 and 28.3 +/- 2.2 mumoles toluene/mgprotein-hr, and half-saturation constants (Ks) of 0.8 +/- 0.5 and 11.5 +/- 2.4 microM, respectively. Strain T102 has a higher capacity than strain T101 to degrade toluene, and kinetic calculations suggest that strain T102 may be a major contributor to toluene biodegradation in the stream.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=10188284&dopt=Abstract

Due to the complexity , the biological process of hair growth is still a work in progress. Nonetheless, several therapeutic methods including prescription medications, transplant surgery, nutritional suppelements, and even snake oils have been in use to help those who attempt to restore their hair. None of these approaches are perfect due to the heterogeneity in the causes that underlie hair loss. Unfortunately, most of these chemical drugs and hair transplantation operations are accompanied by undesirable side effects.

Hair Million of Dream Pharm provides an alternative approach to hair loss problems. Numerous anecdotal cases have demonstrated that this herbal formula based on the authentic Chinese herbs from Chinese Pharmacopoeia actually improves the age-related hair thinning and hair loss among a significant fraction of people who take it as suggested. We still do not understand the mechanisms of action as to how Hair Million works to stop hair loss and promote hair growth, despite all the positive anecdotal demonstration. Neither scientific research nor placebo controlled clinical analysis has been conducted due to the high cost of such trials. Lack of scientific/clinical research is quite common in herbal arena. Just because science hasn't scrutinized doesn't mean we should stop taking daily food and herbal supplements altogether: our life must go on until we have better understandings of food and herb that we have been taking generation after generation. There are two merits in this hair restoration herbal formula: Firstly, Hair Million is relatively inexpensive compared with other methods, and secondly, it is made of edible herbs that are known to be safe when consumed in regular quantities.

DHEA is a natural hormone, and it is produced in our body by the adrenal glands. DHEA has been suggested to provide numerous potential benefits. DHEA (or dehydroepiandrosterone) is converted into androgens (male hormones) or estrogens (female hormones) in the cells.

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