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Pathogen research abs 1 || Pathogen research abs 2 || Pathogen research abs 3 || Pathogen research abs 4 || Pathogen research abs 5 || Hormone and endocrine research abs 1 || Hormone and endocrine research abs 2 || Hormone and endocrine research abs 3 || Hormone and endocrine research abs 4 || Hormone and endocrine research abs 5

Anticancer Res. 1999 Jan-Feb;19(1B):779-87.
Chronological transition of the age-adjusted incidence rates (AAIRs) of 20 major neoplasias from early 1960s to mid-1980s.

Kodama M, Murakami M, Kodama T.

Kodama Research Institute of Preventive Medicine, Nagoya, Japan.

The purpose of this study was to investigate the chronological changes in the age-adjusted incidence rates (AAIRs) of cancers of all sites as well as of 20 major cancers worldwide, which were tested using 5 consecutive IARC Data books from the early 1960s to mid-1980s. We used log-transformed AAIR data (log AAIR) for the statistical analysis of group data. For the follow-up study of a given neoplasia, we prepared 5 world populations using 5 international Data books, and calculated the mean and standard deviation of log AAIR for each tumor, for each sex and for each of 5 world populations for the follow-up study of cancer risks of 20 major cancers as well as cancers of all sites. The results obtained are as follows: a) the log AAIR of cancers of all sites, as compared between the early 1960s group and the mid-1980s group, significantly increased in both the male population (+32.1%) and the female population (+18.0%). b) The remarkable decrease of risks (log AAIRs) for cancers of the stomach and the uterine cervix (non-Western type cancers) and a remarkable increase of risks for cancers of the lung of both sexes, and of the female breast (Western type cancers), were observed from the early 1960s to the mid-1980s--a finding to indicate that the Westernization influences the cancer risk world-wide. c) The follow-up study of cancer risks of individual tumors in both individual population units and the world populations revealed the presence of a number of cancer risk changes that do not fit the definition of "Westernization of cancer risk pattern": i) an abrupt and remarkable increase of skin cancer risk that took place in both the East and the West in the time range of early 1960s to early 1970s; ii) a remarkable increase of liver cancer risk in both the East and the West in the early 1960s to mid-1980s; iii) sex-discriminative cancer risk increase, as exemplified by the contrast between significant risk increase of male sex organs (the prostate and the testis) and complete lack of risk increase in the female sex organs (the uterus and the ovary). d) Evidence was presented to suggest the possibility that some environmental hormone(s) may be implicated in the above changes in cancer risk that do not fit the definition of the "Westernization of cancer risk pattern". e) The importance of interdisciplinary exploration was stressed when assessing the scope of environmental hazards for both the risk of cancers of all sites and the reproductive activity of the human being.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=10216493&dopt=Abstract

Ugeskr Laeger. 2003 May 12;165(20):2085-8.
[Medullary compression in men with prostatic cancer. A retrospective study of medical records]

[Article in Danish]

Klarskov N.

Urologisk Sektion, Amtssygehuset i Glostrup. Niels_klarsko-box.dk

INTRODUCTION: The aim of the study was to describe symptomatology and prognosis in patients with spinal cord compression and prostate cancer. MATERIAL AND METHODS: The material was retrospective and included patients from 1.1.1996 to 31.12.2000 with compression of the medulla, dura or intraspinal roots. Data collection was concluded on 30.6.2001. RESULTS: Seven patients had the cancer diagnosis at the time of the intraspinal compression. The most frequent debut-symptom was acceleration of pain, two thirds had neuropathic pain, and three patients were free of pain. Paresis was present in 23 patients and 18 had sensory symptoms. External irradiation was given to 29 patients, three had a neurosurgical procedure and irradiation and two patients had just endocrine manipulation. The treatment result was good or moderately good on pain in three fourths of the patients, and on the neurological symptoms in two thirds. Eight patients had recurrence. Median survival was three months for patients with hormone resistant progression and two years for those patients, who got their cancer diagnosis in connection with the intraspinal compression. CONCLUSION: Rapid acceleration of pain is an early symptom of intraspinal compression. Morphine-resistant pain or neuropathic pain and neurologic symptoms are typical findings, but none of the symptoms are obligatory. Early acute treatment may give good recovery and the survival is comparable with the survival of patients whose illnesses are in the same stadium but who do not suffer from intraspinal compression.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12812097&dopt=Abstract

Actas Urol Esp. 2003 Mar;27(3):221-5.
[Adrenal ganglioneuroma. Report of a new case]

[Article in Spanish]

Arredondo Martinez F, Soto Delgado M, Benavente Fernandez A, Basquero Gonzalez B, Zurera Cosano A, Linares Armada R.

Servicio de Urologia, Hospital Infanta Elena, Huelva.

Ganglioneuroma is a benign neoplasm arising from neural crest tissue and is composed of mature ganglion cells and Schwann's cells. Most ganglioneuromas arise in the posterior mediastinum followed by the retroperitoneum. Only a small proportion of ganglioneuromas are adrenal in origin and occur most commonly in children and young adults. Characteristically, ganglioneuromas do not secrete excess catecholamines or steroid hormones, and they are usually clinically silent lesions incidentally detected in imaging studies for unrelated reasons. Because it is an uncommon lesion, adrenal ganglioneuromas is not included in the differential diagnosis of adrenal incidentalomas in adults. We presents a new case of a 6.5 cm adrenal ganglioneuroma incidentally diagnosed in a 50-year old male patient. The exeresis of the adrenal mass and pathohistological study confirmed the diagnosis.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12812120&dopt=Abstract

Clin Chem Lab Med. 2003 May;41(5):686-92.
Influence of gender on circulating cardiac natriuretic hormones in patients with heart failure.

Emdin M, Passino C, Del Ry S, Prontera C, Galetta F, Clerico A.

Department of Cardiovascular Endocrinology, CNR Institute of Clinical Physiology, University of Pisa, Pisa, Italy.

In order to study the influence of gender on circulating levels of cardiac natriuretic hormones (CNHs) in heart failure, we measured the plasma levels of atrial natriuretic peptide (ANP) and brain natriuretic peptide (BNP) by means of highly sensitive and specific IRMA methods in 239 consecutive patients (age 64.7 +/- 11.6 years, range 21-89 years; 170 men and 69 women) with cardiomyopathy. There was different response of CNH according to gender in patients with heart failure, as indicated by the ratio between the individual CNH values of patients and the gender-specific cut-off values. Indeed, the mean ratio for ANP found in men (3.6 +/- 3.6) was significantly higher (p = 0.0075) than that found in women (2.4 +/- 2.1). The mean ratio for BNP was on average 2.3 fold higher (15.9 +/- 27.1 in men and 6.9 +/- 6.8 in women, p = 0.0084). Moreover, age, ejection fraction, and disease severity independently and significantly contributed to regression with both ANP (R = 0.612, F = 39.969, p < 0.0001) and BNP (R = 0.656, F = 49.957, p < 0.0001) values, while gender did not. In conclusion, our study suggests a different, gender-specific activation of the CNH system in this clinical condition, although age, ejection fraction and disease severity seem to be more powerful predictors than gender of circulating levels of ANP and BNP in patients with heart failure.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12812268&dopt=Abstract [PubMed - in process]

Pituitary. 2002;5(3):163-8.
Adrenocorticotrope deficiency with clinical evidence for late onset in combined pituitary hormone deficiency caused by a homozygous 301-302delAG mutation of the PROP1 gene.

Lamesch C, Neumann S, Pfaffle R, Kiess W, Paschke R.

III. Medical Department, University of Leipzig, Germany.

Combined pituitary hormone deficiency (CPHD) can be caused by mutation of the pituitary transcription factors POU1F1 or PROP1. More recently mutations in the HESX1, the LHX3 and LHX4 transcription factor genes have also been described as a cause in patients with CPHD. In most patients the disorder is characterized by an impaired production of GH, TSH, PRL and gonadotropins. In some cases of CPHD adrenocorticotropin deficiency is also present. We report the progressive CPHD and its molecular etiology in a woman with CPHD presenting with first symptoms of ACTH/cortisol deficiency at the age of 48 years. The 49 year old patient's initial symptoms were growth retardation at the age of 2 years and symptoms of hypothyroidism at the age of 5 years. The patient never entered puberty spontaneously. No familial history of delayed puberty, growth retardation or other symptoms of CPHD were present. At the age of 48 years the patient presented with the first symptoms of hypocortisolism such as recurring hypoglycaemias and hyponatriaemia with coma. Cortisol, ACTH, TSH, fT3, fT4 and GH as well as LH, FSH and PRL were measured in basal conditions. GH, cortisol and ACTH were also measured in response to an Insulin Tolerance Test. Molecular analysis was performed by PCR amplification and sequencing of exon 1-3 of the PROP1 gene. The patient had insufficiencies of TSH, LH, FSH and GH. PRL was normal. Serum cortisol was low and basal ACTH was normal. However, there were no responses of cortisol, ACTH and GH to hypoglycaemia. Magnetic resonance imaging showed a hypoplastic anterior pituitary lobe. Direct sequencing revealed a homozygous 2 base-pair deletion 301-302delAG in exon 2 of the PROP1 gene. This case suggests that in patients with CPHD ACTH producing cells may be involved at a rather late age.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12812307&dopt=Abstract

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