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Lutein-20||Herbs for headache, fever, and migraine ||
Milk thistle||Saw palmetto||
Triple B Super Vision||Garlic, Ginger, and Grapeseed Extract||
Ginseng and Ginkgo||Hair Million||
DHEA||Coenzyme Q10||
Sleep Aid herbal formula - natural sleep aid||Herbal Breath - herbs for bad breath problems.||
Weight loss herbal formula for menopause and pms||Ginkgo biloba||
Colon cleansing, Laxative||ViaVita, Lecithin for healthy liver
Fatty acids resources:
Pathogen research abs 1 || Pathogen research abs 2 || Pathogen research abs 3 || Pathogen research abs 4 || Pathogen research abs 5 ||
Hormone and endocrine research abs 1 || Hormone and endocrine research abs 2 || Hormone and endocrine research abs 3 || Hormone and endocrine research abs 4 || Hormone and endocrine research abs 5
Endokrynol Diabetol Chor Przemiany Materii Wieku Rozw. 1998;4(1):13-7.
[The thyroid function in children with viral meningitis]
[Article in Polish]
Szychowska Z, Kucharska W.
Klinika Chorob Zakaznych Wieku Dzieciecego AM we Wroclawiu.
Thyroid function was investigated in children affected with viral meningitis caused by Mumpsvirus or enteroviruses. Serum or plasma levels of thyroid-stimulating hormone (TSH), triiodothyronine (T3), free triiodothyronine (FT3), thyroxine (T4) and free thyroxine (FT4) were measured twice in course of the disease: at admission and at recovery (day 10-14 from the onset of illness). The levels of hormones were measured by radioimmunoassay (RIA) or by enzyme linked fluorescent assay (ELFA). A decrease in serum or plasma concentrations of TSH, T3, FT3 and T4 (T4 - only when measured by RIA) was found at the beginning of illness as compared to the controls, which indicates the low-T3 syndrome in children with viral meningitis. These disturbances were present also at recovery. When comparing thyroid function in children suffering from bacterial and viral meningitis, a more significant decrease in the levels of thyroid hormones (especially T3 and FT3) was found at the beginning of bacterial than viral meningitis. Un resolved questions are the causes and the importance of low-T3 syndrome in children with viral meningitis.
online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12818109&dopt=Abstract [PubMed]
Endokrynol Diabetol Chor Przemiany Materii Wieku Rozw. 1998;4(1):19-25.
[The thyroid function in children with purulent meningitis]
[Article in Polish]
Szychowska Z, Kucharska W.
Klinika Chorob Zakaznych Wieku Dzieciecego AM we Wroclawiu.
Purulent meningitis (BM) is a complex process and its outcome is largely influenced by host's response, both inflammatory or endocrine, to the infection. In this study thyroid function in children with BM was investigated by measuring serum or plasma levels of thyroid-stimulating hormone (TSH), triiodothyronine (T3), free triiodothyronine (FT3), thyroxine (T4) and free thyroxine (FT4) 3 times in course of the disease: at admission, after 24-48 hrs. of treatment and at recovery (day 10-14 from the onset of the disease). The levels of hormones were measured by radioimmunoassay (RIA) or by enzyme linked fluorescent assay (ELFA). A decrease in serum or plasma concentrations of all measured hormones was found at the beginning of BM as compared to the controls. This indicates the low T4 variant of sick euthyroid syndrome in children with BM, which usually occurs in seriously ill patients. The levels of measured hormones rose at recovery in comparison to their initial values, but T3 concentrations and TSH (TSH - measured by RIA) were still lower in comparison to the controls. Serum T3 concentrations were significantly lower in children with sequelae of BM than in children who recovered without sequelae. This indicates a possible adverse effect of disturbed thyroid function resulting in low-T3 syndrome on the outcome from BM. An adjunctive anti-inflammatory treatment with dexamethasone started 10-15 minutes before the first antibiotic dose and, given every 12 hrs in a dose of 0.4 mg/kg for 2 days, it resulted in greater decrease in T3, FT3, T4 and FT4 plasma levels in comparison to non-steroid treated children.
online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12818110&dopt=Abstract [PubMed]
Endokrynol Diabetol Chor Przemiany Materii Wieku Rozw. 1998;4(1):45-53.
[The analysis of exercise efficiency and autonomic function in children with nontoxic nodular goiter during suppressive L-thyroxine therapy]
[Article in Polish]
Bossowski A, Gardziejczyk M, Urban M.
II Klinika Chorob Dzieci AM w Bialymstoku.
The aim of the study was the analysis of exercise efficiency and the function of the autonomic system in children with a nontoxic nodular goiter (NNG) subjected to TSH suppression. The study comprised 17 children, aged 11-18 years, with NNG treated with suppressive L-thyroxine doses for 6-36 months (mean 24 months). Exercise efficiency was examined on a moving track according to the protocol of Bruce. Analysis of the autonomic function was based on functional tests of the circulatory system and monitoring of the QTc interval. A group of healthy children with a negative heart and thyroid history served as control. Statistically significant acceleration of the heart rate (p<0.01) was found in NNG patients subjected to TSH suppression, compared with controls. The remaining results included a fall in exercise efficiency parameters:work load (p<0.001) and duration (p<0.003), metabolic equivalent (p<0,005), and the maximal oxygen consumption index Vo2 (p<0.005) at normal values of double product. A significant decrease was revealed in the ejection fraction during exercise (61+/-1.9 vs 67+/-5.9 p<0.001) in the group of children with nontoxic nodular goiter. Conclusions: 1. In the subclinical state of hyperthyroidism no functional disorders were observed in the autonomic system, which controls the circulatory system. 2. Exercise tolerance was reduced in patients with nontoxic nodular goiter subjected to TSH suppression. 3. A relationship was found between the level of thyroid hormones (TSH, FT4) indicating the subclinical state of hyperthyroidism and the acceleration of heart rate, reduction in the ejection fraction of the left ventricle of the heart after exercise and shortening of load test duration. 4. Higher rest values of heart rate were observed in children treated with suppressive doses of L-thyroxine, compared with controls.
online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12818113&dopt=Abstract [PubMed]
Endokrynol Diabetol Chor Przemiany Materii Wieku Rozw. 1998;4(1):61-6.
[Effect of recombinant human growth hormone treatment on glucose metabolism]
[Article in Polish]
Bodalski J, Mianowska B.
II Klinika Chorob Dzieci Instytutu Pediatrii AM w Lodzi.
Deciding to administer human growth hormone (hGH, somatotrophin) physicians often consider the possibility of adverse reactions of such a treatment, and among the others, they have regard to its effects on glucose metabolism. The problem is particularly important in patients treated with higher than physiological doses of hGH (children with constitutional short stature, girls with Turner's syndrome). Recent studies suggest, that although the effect of hGH on glucose metabolism is undeniable, replacement doses of this hormone practically do not threaten with diabetes development, but its higher than physiological doses can impair insulin secretion and, in exceptional cases, cause transient diabetes.
online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12818115&dopt=Abstract [PubMed]
J Biol Chem. 1999 Apr 30;274(18):12431-7.
Glucocorticoid induction of epithelial sodium channel expression in lung and renal epithelia occurs via trans-activation of a hormone response element in the 5'-flanking region of the human epithelial sodium channel alpha subunit gene.
Sayegh R, Auerbach SD, Li X, Loftus RW, Husted RF, Stokes JB, Thomas CP.
Department of Internal Medicine, University of Iowa College of Medicine, Iowa City, Iowa 52246, USA.
In airway and renal epithelia, the glucocorticoid-mediated stimulation of amiloride-sensitive Na+ transport is associated with increased expression of the epithelial Na+ channel alpha subunit (alphaENaC). In H441 lung cells, 100 nM dexamethasone increases amiloride-sensitive short-circuit current (3.3 microA/cm2 to 7.5 microA/cm2), correlating with a 5-fold increase in alphaENaC mRNA expression that could be blocked by actinomycin D. To explore transcriptional regulation of alphaENaC, the human alphaENaC 5'-flanking region was cloned and tested in H441 cells. By deletion analysis, a approximately 150-base pair region 5' to the upstream promoter was identified that, when stimulated with 100 nM dexamethasone, increased luciferase expression 15-fold. This region, which contains two imperfect GREs, also functioned when coupled to a heterologous promoter. When individually tested, only the downstream GRE functioned in cis and bound GR in a gel mobility shift assay. In the M-1 collecting duct line Na+ transport, malphaENaC expression and luciferase expression from alphaENaC genomic fragments were also increased by 100 nM dexamethasone. In a colonic cell line, HT29, trans-activation via a heterologously expressed glucocorticoid receptor restored glucocorticoid-stimulated alphaENaC gene transcription. We conclude that glucocorticoids stimulate alphaENaC expression in kidney and lung via activation of a hormone response element in the 5'-flanking region of halphaENaC and this response, in part, is the likely basis for the up-regulation of Na+ transport in these sites.
online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=10212217&dopt=Abstract
The average human scalp is covered by approximatey 100,000 hair follicles. Each hair undergoes
hair cycle and normally 50-100 hairs randomly fall out a day, which is unnoticeable because lost hair is replaced by as many new hairs springing up daily. Hair loss results from the fall out of hair from the hair follicle. Alopecia or excessive, premature hair loss is the condition caused by many factors.
Loss of hair itself does not pose critical health problems because biological role of human hair is relatively marginal. Hair on our scalp protects the head from mechanical shock, heat loss, and exposure to UV-light. The eyelashes and eyebrowes protect the eyes, and hair in the ear canal or the nasal passages help filter out particles and pathogens, thus protecting our internal organs.
However, hair does play important social role: it is one of the major determinants of our appearance and identity in daily life. Fullness of hair also implicates or manifests physical integrity and youthfulness of the person. Losing hair could have more than just emotional impacts on individuals.
The hair is a unique organ that goes through a characteristic cycle consisting of an immature phase, a growing phase called anagen, a transitional phase between the growing phase and the resting phase called catagen, and finally a resting phase called telogen in which the hair stops growing, waiting to fall out. 85-90% of hairs on our body are in anagen phase or growing phase, which lasts anywhere from two to five years. This phase is followed by a short regression phase, or catagen, which lasts 2-3 weeks. Approximately 1% of hair follicles are in catagen. Approximately 10-15% of hair follicles are in the resting phase, the telogen, which lasts about 3-5 months. Hair follicles typically goes through 10-20 asynchronous cycles during the lifetime.
Persistent loss of more than 150 hairs would consist a state of hair loss, or alopecia, albeit it could be temporary.
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Lutein ||
Progesterone Cream ||
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