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Pathogen research abs 1 || Pathogen research abs 2 || Pathogen research abs 3 || Pathogen research abs 4 || Pathogen research abs 5 || Hormone and endocrine research abs 1 || Hormone and endocrine research abs 2 || Hormone and endocrine research abs 3 || Hormone and endocrine research abs 4 || Hormone and endocrine research abs 5





Ann Intern Med. 1999 Feb 16;130(4 Pt 1):270-7.
Elevated serum estradiol and testosterone concentrations are associated with a high risk for breast cancer. Study of Osteoporotic Fractures Research Group.

Cauley JA, Lucas FL, Kuller LH, Stone K, Browner W, Cummings SR.

Department of Epidemiology, Graduate School of Public Health, University of Pittsburgh, Pennsylvania 15261, USA.

BACKGROUND: The relation between endogenous steroid hormones and risk for breast cancer is uncertain. Measurement of sex hormone levels may identify women at high risk for breast cancer who should consider preventive therapies. OBJECTIVE: To test the hypothesis that serum concentrations of estradiol and testosterone predict risk for breast cancer. DESIGN: Prospective case-cohort study. SETTING: Four clinical centers in the United States. PARTICIPANTS: 97 women with confirmed incident breast cancer and 244 randomly selected controls; all women were white, 65 years of age or older, and were not receiving estrogen. MEASUREMENTS: Sex-steroid hormone concentrations were assayed by using serum that was collected at baseline and stored at -190 degrees C. Risk factors for breast cancer were ascertained by questionnaire. Incident cases of breast cancer were confirmed by review of medical records during an average period of 3.2 years. RESULTS: The relative risk for breast cancer in women with the highest concentration of bioavailable estradiol (> or = 6.83 pmol/L or 1.9 pg/mL) was 3.6 (95% CI, 1.3 to 10.0) compared with women with the lowest concentration. The risk for breast cancer in women with the highest concentration of free testosterone compared with those with the lowest concentration was 3.3 (CI, 1.1 to 10.3). The estimated incidence of breast cancer per 1000 person-years was 0.4 (CI, 0.0 to 1.3) in women with the lowest levels of bioavailable estradiol and free testosterone compared with 6.5 (CI, 2.7 to 10.3) in women with the highest concentrations of these hormones. Traditional risk factors for breast cancer were similar in case-patients and controls. Adjustments for these risk factors had little effect on the results. CONCLUSIONS: Estradiol and testosterone levels may play important roles in the development of breast cancer in older women. A single measurement of bioavailable estradiol and free testosterone may be used to estimate a woman's risk for breast cancer. Women identified as being at high risk for breast cancer as determined by these hormone levels may benefit from antiestrogen treatment for primary prevention.


online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=10068384&dopt=Abstract



Gen Comp Endocrinol. 1999 Mar;113(3):464-77.
Effects of photoperiod on the cessation of growth during autumn in male red deer and growth hormone and insulin-like growth factor-I secretion.

Webster JR, Corson ID, Littlejohn RP, Stuart SK, Suttie JM.

AgResearch, Invermay Agricultural Centre, Mosgiel, New Zealand.

Male red deer undergo seasonal cycles of food intake and growth rate, which are high during spring and low during winter, despite high quality food ad libitum. Hormonal profiles during the cessation of growth in autumn and the potential role of photoperiod in the timing of the observed changes have been investigated. Whether this seasonal decrease in growth affected the response of GH and IGF-I to fasting was also examined. Two groups of six male 1-year-old red deer were exposed to different photoperiods after the summer solstice. One group (C) was given a simulated natural photoperiod while the other group (SS) was maintained on a summer solstice photoperiod (16L:8D). GH was measured in blood collected continuously and divided into pools every 5 min for 24 h in the fed state and after a 48-h fast on two occasions; the first was in November before photoperiod manipulation began and the second was in April approximately 16 weeks after initiating treatments. IGF-I, prolactin, and testosterone were measured in weekly samples. Individual live weight and group food intake were also measured each week. The normal growth pattern seen in the C group was delayed in the SS group. Thus, from 7 March until the second GH sampling on 11 April the live weight of deer in group C fell; in contrast, deer in group SS continued to grow (-43 vs 186 g/day s.e.d. = 65.5, P < 0. 01). Food intake changes reflected the pattern of growth in both groups. Mean GH (P < 0.05), GH pulse amplitude (P < 0.01), and IGF-I (P < 0.001) declined in both groups from November to April. This decline was more marked in group C and in April these parameters were all lower in group C than in group SS (GH, P < 0.05; IGF-1, P < 0.01). Prolactin levels in April were also lower in group C than in group SS (P < 0.01); testosterone was not affected by treatment. Fasting increased mean GH and GH pulse amplitude in both groups in November (P < 0.05). In April, the fasting response differed between the groups. In group C, mean GH, pulse amplitude, and pulse frequency were all greater in the fasted state than in the fed state (P < 0.05), while in group SS there were no significant differences (P > 0.05). IGF-I was lower in the fasted state than in the fed state at both sampling dates (P < 0.001). The seasonal decline in food intake and growth is associated with decreased GH, IGF-I, and prolactin concentrations, and increased testosterone and the GH response associated with fasting. All these changes except those of testosterone were delayed or reduced by continued exposure to a summer solstice photoperiod in autumn. The decreased photoperiod in autumn may thus influence the normal timing of the seasonal growth cycle. 1999 Academic Press.


online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=10068507&dopt=Abstract



J Cardiovasc Risk. 2003 Feb;10(1):57-60.
Influence of hormone replacement therapy on C-reactive protein: population-based data.

Primatesta P, Falaschetti E, Poulter NR.

Department of Epidemiology and Public Health, Royal Free and University College Medical School, London WC1E 6BT, UK. paolaublic-health.ucl.ac.uk

BACKGROUND: Observational data, possibly resulting from confounding due to self-selection, show that use of hormone replacement therapy (HRT) is associated with reduced coronary risk, while randomized trial data do not. One possible explanation for the trial findings is that adverse effects of HRT-- such as an elevation of C-reactive protein (CRP)--might counterbalance other benefits of HRT. DESIGN: Women aged 40-74 years were identified from a cross-sectional, annual nationally representative study of the English population, the Health Survey for England 1998. Valid data were available from 4,112 of the 4,319 women in this age group. Of these, 710 (17%) were current, 465 (11%) past and 2,937 (71%) never users of HRT. METHODS: To study the association between current and past use of HRT and CRP levels. CRP was expressed as a continuous variable (logged) and in percentiles. RESULTS: Current, past and never users of HRT did not differ in terms of age, body mass index or smoking status. However, compared with past and never users of HRT, current users tended to take more vigorous physical activity and were more likely to drink more alcohol than is currently recommended. Excluding potentially confounding conditions, median CRP levels were significantly higher in current (2.5 mg/l) and in past (1.9 mg/l) than never HRT users (1.4 mg/l). Controlling for age, smoking and body mass index logged CRP remained significantly raised only among current users. CONCLUSIONS: HRT use was significantly and independently associated with raised CRP levels in an English nationally representative sample. Increases in CRP may represent one plausible mechanism by which HRT may adversely affect risk of coronary heart disease.


online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12569237&dopt=Abstract



Gen Comp Endocrinol. 1999 Mar;113(3):343-59.
Characterization of thyroid hormone 5'-monodeiodinase activity in the turtle (Trachemys scripta).

Hugenberger JL, Licht P.

Department of Integrative Biology, University of California, 3060 Valley Life Science Building, Berkeley, California, 94720-3140, USA.

Thyroid hormone metabolism by 5'-monodeiodinase enzymes (5'MD) was characterized in peripheral tissues of the turtle, Trachemys scripta, and compared with activity measured in the rat. Based on differences in pH dependence, sensitivity to inhibitors, substrate affinity, and cofactor requirements, at least two types of enzyme activities have been identified in the turtle. A 5'MD activity was measured in liver and kidney microsomal fractions that exhibits inhibition by 2n-propyl-6-thiouracil (PTU), a higher affinity for rT3 (Km = 2 microM) than for T4 (Km = 6.5 microM), a low cofactor dependence, and a high pH optimum for T4 metabolism. The characteristics of this turtle low affinity T4 activity correspond to the mammalian type I monodeiodinase. A second type of monodeiodinase (MD) activity that is less sensitive to PTU, has a higher affinity for T4 (Km = 1 nM), a higher cofactor requirement, and a lower pH optimum was colocalized with the first form. Both turtle MD activities remain active over a range of temperatures, allowing for activity at the preferred body temperature of this species (28 to 37 degrees C compared to the 37 degrees C optimum in the rat). Based on limited comparative data of MD systems from several fish and birds, the turtle most closely resembles avian species. Like birds, turtles possess a mammalian-like type I activity and have colocalized MD forms in the liver. However, the second turtle MD form (MDH) is not comparable to the mammalian or avian MDII-like activity. Analysis of the deiodinase products from both turtle MDs by high-performance liquid chromatography confirmed that the putative turtle MDI produces T3 from T4 as expected. The MDH produces rT3 from T4 as does the mammalian type III form, but MDH has a wider tissue distribution (kidney, liver, pancreas, heart, ovary, and brain) and distinct enzyme kinetics. Moreover, MDH activity in the turtle kidney is 100-fold higher than in the liver, indicating that the kidney may play a critical role in the metabolism of thyroid hormones in the turtle; this high renal activity distinguishes the turtle from all other vertebrates studied. 1999 Academic Press.


online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=10068496&dopt=Abstract



East Afr Med J. 1998 Nov;75(11):644-6.
Female breast cancer with 'clandestine' lymph node metastases.

van Bogaert LJ.

Department of Obstetrics and Gynaecology, Faculty of Medicine and Health Sciences, University of Transkei Umtata, South Africa.

OBJECTIVE: To determine whether or not breast cancer with 'clandestine' lymph node metastases differ in any important way from breast cancer with macrometastases. METHODS: A study of 116 female breast cancers over a period of two years, whose axillary contents were immuno-stained prospectively to detect the possible presence of clandestine metastases, that is tumour cells identifiable only after monoclonal antibody immunostaining. RESULTS: The prevalence of 'clandestine' lymph node metastases was 14.7%. The maximum number of 'clandestine' lymph node metastases was three per axilla. In the cases with 'clandestine' metastases the infiltrating lobular carcinoma (ILC) type was more frequently seen than in the node negative cases: (p = 0.038) (RR = 2.44; OR = 4.0). There was no difference in the hormone receptor status between the cases without lymph node metastases and with lymph node metastases, 'clandestine' or macrometastases. CONCLUSION: No significant difference was found between breast cancers with 'clandestine' or with macrometastases with regard to their clinico-pathologic features and hormone receptor status. The detection of 'clandestine' metastases is a painstaking and time-consuming procedure which, so far, has not been shown to be of clinical significance.


online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=10065176&dopt=Abstract







Sudden, and premature hair loss and baldness is a problem in many ways. Baldness is indeed becoming an increasing concern in the current aging society.
It changes personal appearance and identity in social context. Saw palmetto berry extract is a widely known herbfor hair loss as well as BPH problems in Western world. Saw palmetto berry contains phytochemicals that inhibits 5-alpha-reductase that converts testosterone to DHT.

There are a number of traditional herbs that could stop hair loss and promotes hair growth. Numerous personal experiences and anecdotal cases testify that the herbal formula based on the Chinese herbs improves the situation of the age-related hair thinning and hair loss for a large fraction of people taking it regularly. It is unknown how Hair Million herbs stop hair loss, and promote hair growth due to the lack of scientific research and placebo controlled clinical trials.












DHEA is a natural hormone, and it is produced in our body by the adrenal glands. DHEA has been suggested to provide numerous potential benefits. DHEA (or dehydroepiandrosterone) is converted into androgens (male hormones) or estrogens (female hormones) in the cells. Our bodies produce decreasing amount of DHEA as we get older. various health benefits: To deter aging, improve sexual function/erectile dysfunction, treat cognitive decline, enhance athletic performance, facilitate weight loss, improve strength, prevent osteoporosis, enhance immunomodulation for rheumatic conditions, and treat depression.





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