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Fatty acids resources:

Pathogen research abs 1 || Pathogen research abs 2 || Pathogen research abs 3 || Pathogen research abs 4 || Pathogen research abs 5 || Hormone and endocrine research abs 1 || Hormone and endocrine research abs 2 || Hormone and endocrine research abs 3 || Hormone and endocrine research abs 4 || Hormone and endocrine research abs 5







Ginecol Obstet Mex. 2003 Feb;71:55-9.
[In Process Citation]

[Article in Spanish]

Romero Gutierrez G, Baron Vazquez S, Avecilla Hernandez AA, Ponce de Leon AL.

Instituto Mexicano del Seguro Social, Unidad de Investigacion en Epidemiologia Clinica, Hospital de Gineco-Pediatria No. 48, Leon, Guanajuato.

OBJECTIVE: To assess the prevalence of unsuspected molar pregnancy in patients with first trimester spontaneous abortions. METHODS: An observational, cross-section study was carried out in 396 consecutive patients with diagnosis of first trimester spontaneous abortions, without clinical or ultrasonographic findings suggesting hydatidiform mole, a histological diagnosis was made from the curettage specimens, the cases with diagnosis of any trophoblastic disease prior of curettage were not included in the study. RESULTS: Diagnosis of molar pregnancy was made in 48 out of 396 patients (12.1%). There was not statistical difference in the sociodemographic variables and risk factors analyzed: patient age, familial incomes, years in scholar courses, prior pregnancies, deliveries, spontaneous abortions, number of sons, and prior hormone contraceptive use between the patients with diagnosed molar pregnancy and those patients without the diagnosis of molar pregnancy. CONCLUSION: The prevalence of unsuspected molar pregnancy found in our study (12.1%) was high and the suspicion in these cases according the risk factors of the disease could be difficult, therefore to rule out systematically molar pregnancy in each case of spontaneous abortions is recommended in order to avoid the complications associated with hydatidiform mole.


online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12708351&dopt=Abstract [PubMed - in process]



Ann Endocrinol (Paris). 1979;40(6):545-6.
[Familial case of male pseudohermaphroditism due to 17-ketoreductase defect: late diagnosis in the "aunt" of a patient with the same defect (author's transl)]

[Article in French]

Forest MG, de Peretti E, Campo-Paysaa A.

A 42 year old (46 XY) subject with 17-ketosteroid reductase deficiency was investigated. The patient reared as female, has developed masculine features (facial hair, male distribution of body hair, male body habitus, acne and clitoridomegaly) at about 15 years of age but never consulted. She married at 22 years and for 20 years thought to have a "normal" female sex life. Only when her 14 year old "niece" was investigated (1) and treated for similar problems she realized hers. She had a small phallus with perineal urethra, vaginal pouch absence of labia minora and undescended testis, no breast development. Baseline peripheral studies showed plasma testosterone (T) in the range of Tanner II stade of puberty (150 ng/dl), elevated delta 4-androstenedione (delta 4) (930 ng/dl) and estrone (E1) (33,5 NG/DL) LEVELS 6--8 times above normal, but subnormal estradiol levels. Increased basal gonadotropins showed an hyper-response to LHRH stimulation. Dynamic tests (ACTH test, Dexamethasone suppression, and hCG stimulation) showed that abnormal delta 4 and E1 were not of adrenal origin. In the spermatic veins delta 4 levels were extremely high (239 micrograms/dl) but T levels low (11.4 micrograms/dl). delta 4/T ratio in the spermatic vein was much higher than in the peripheral blood suggesting intact peripheral conversion of delta 4 to T. After castration all hormone levels returned to the range usually seen in agonadic male or female adults.


online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=121222&dopt=Abstract



C R Seances Acad Sci D. 1979 Nov 19;289(13):943-6.
[Gonadostatin and gonadocrinin, polypeptides of ovarian origin with hypophysiotropic activity]

[Article in French]

Ying SY, Guillemin R.

Crude acetic acid extract of Rat ovaries pretreated with pregnant mare serum (PMSG) contains native peptides with two types of separable biological activities: one, molecular weight greater than 10,000 dalton inhibits the secretion of both LH and FSH as stimulated by luteinizing hormone releasing factor (LRF) in a pituitary monolayer culture system and is referred to as gonadostatin; the other, less than 3,500 dalton, stimulates the secretion of gonadotropins and is designated as gonadocrinin. The biological activities of ovarian gonadocrinin can be competitively inhibited by an LRF-analog-antagonist, D-Phe2, D-Trp6-LRF. These ovarian peptides may participate in physiological control of pituitary LH/FSH secretion.


online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=121254&dopt=Abstract



Jugosl Ginekol Opstet. 1979;19(1-2):45-57.
[Importance of determining gonadotropin levels in the serum in secondary amenorrhea]

[Article in Serbo-Croatian (Roman)]

Bila S, Kraincanic M, Sulovic V, Genbacev O, Matijasevic S.

On the basis of the results of the analysis of FSH, LH, and prolactin values in the serum of patients with secondary ammenorrhea, four groups of patients were formed: group 1 with low values of both gonadotropin hormones, group 2 with low FSH and high LH values, group 3 with high FSH and high LH values, and group 4 with the basdal gonadotropin values within normal. The use of functional tests proved helpful in the differentiation of the causes of amenorrhea. The use of the LH-RH test is of particular significance in the differentiation of the degree of changes in patients from group 1 and 4. To determine adequate therapy, the determination of estrogen in patients from group 1 and 3 is imperative. A successful treatment of secondary amenorrhea depends on its duration and a timely detection of its causes.


online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=121593&dopt=Abstract



Cancer Sci. 2003 Jan;94(1):103-11.
Up-regulation and nuclear localization of beta-catenin in endometrial carcinoma in response to progesterone therapy.

Saegusa M, Hamano M, Kuwata T, Yoshida T, Hashimura M, Akino F, Watanabe J, Kuramoto H, Okayasu I.

Department of Pathology, Kitasato University School of Medicine, 1-15-1 Kitasato, Sagamihara, Kanagawa 228-8555. msaegused.kitasato-u.ac.jp

Ovarian hormones are considered to be capable of regulating expression of beta-catenins. A possible role of beta-catenin in alteration of cell morphology has been proposed, but little is known about beta-catenin expression during changes in the tumor morphology of endometrial carcinomas induced by progesterone therapy. To clarify changes in expression of beta-catenin and their relation to morphological alteration, expression of hormone receptors and several cell kinetic markers, sequential biopsy and hysterectomy specimens of 23 endometrial carcinoma and 6 complex hyperplasia with atypia (atypical hyperplasia) cases receiving progesterone therapy were investigated. In vitro assay was also conducted using two endometrial carcinoma cell lines (HEC265 and Ishikawa) expressing progesterone receptors (PRs). An increase of nuclear beta-catenin accumulation was evident during progesterone therapy in endometrial carcinomas and atypical hyperplasias. The nuclear labeling indices were significantly associated with gene mutations and alteration in morphological features in response to progesterone, independently of the status of Ki-67, p21WAF1 and p27Kip1, and hormone receptors. In HEC265 having a beta-catenin gene mutation (A32V), cytoplasmic beta-catenin levels were elevated by progesterone treatment, linked to down-regulation of PR expression, but such changes were relatively minor in Ishikawa without the gene alterations. These findings demonstrate a possible role of progesterone in regulation of beta-catenin expression in endometrial tumors. Moreover, nuclear beta-catenin accumulation, like gene abnormalities, is associated with the alteration of tumor morphology due to progesterone, indicating that beta-catenin may be a clinically useful marker of hormone therapeutic effects.


online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12708483&dopt=Abstract








Vitamins, amino acids, oils for topical application, and prescription medications...
There are a number of approaches to hair loss problems.
Hair Million is an herbal alternative. It is a formula made of traditional, edible herbs and has been anecdotally demonstrated the efficacy to ward off hair loss problems.

There is no singular medical or alternative cure for hair loss since the biology of hair growth is a highly complicated phenomenon. It is unknown how Hair Million stops hair loss, and promotes hair restoration. The advantages of Hair Million over other approaches are, firstly, Hair Million is comparatively inexpensive, and secondly, it is made only of traditionally used safe and healthy herbs that promote hair growth according to Chinese pharmacopoeia. In addition, Hair Million is cardiotonic, meaning that Hair Million consists of herbs that strengthens your heart, according to Chinese medicine. There is an interesting research paper which correlates baldness to heart diseases: people with alopecia or hair loss problems are significantly more likely to develop heart attacks.














DHEA is a natural hormone, and it is produced in our body by the adrenal glands. DHEA has been suggested to provide numerous potential benefits. DHEA (or dehydroepiandrosterone) is converted into androgens (male hormones) or estrogens (female hormones) in the cells.







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