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Pathogen research abs 1 || Pathogen research abs 2 || Pathogen research abs 3 || Pathogen research abs 4 || Pathogen research abs 5 || Hormone and endocrine research abs 1 || Hormone and endocrine research abs 2 || Hormone and endocrine research abs 3 || Hormone and endocrine research abs 4 || Hormone and endocrine research abs 5

Recent Adv Stud Cardiac Struct Metab. 1975;8:427-36.
The influence of growth hormone on myocardial weight and water fraction in rats.

Powis RL.

While conducting experiments on the role of growth hormone in cardiac hypertrophy, data were also gathered on the influence of growth hormone on myocardial wet and dry weight in relation to body weight and myocardial water fraction. Male rats with either in intact hypophysis or a hypophysectomy were bilaterally adrenalectomized, and corticoids were replaced with deoxycorticosterone and hydrocortisone therapy. Hypophysectomized rats were further treated with thyroxine, testosterone, and, where appropriate, bovine growth hormone. The hormone replacement program re-established systolic blood pressure in hypophysectomized rats to levels observed in intact-hypophysis rats. The left ventricle weight (wet and dry)/body weight ratio was used as an indication of relative myocardial weight. Hypophysectomized rats without growth hormone were observed to have higher left ventricle weight/body ratios than both hypophysectomized rats with growth hormone and intact-hypophysis rats. Further, rats without growth hormone were determined to have a lower myocardial water content than rats with growth hormone. Mathematical analysis established that the relationship between myocardial water fraction and ventricular wet weight is nonlinear. This relationship shows that small changes in water fraction can produce large changes in ventricular wet weight. The reasons for the different water content in the myocardium were not further investigated.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=129837&dopt=Abstract

C R Seances Soc Biol Fil. 1975;169(3 Suppl):778-84.
[Tumor formations and cell proliferation in invertebrates]

[Article in French]

Vago C.

Critical analysis of the present status of invertebrate oncology through an appreciation of data relative to the various origins of tumors : spontaneous formations, cell proliferations induced by viruses, so-called hereditary tumors, effects of injuries induced by carcinogenic agents, irradiations or hormones on the formation of neoplasms in invertebrates. Attempts are made to interpret these data as compared with those obtained in medical and veterinary oncology. The possible direction of studies on invertebrates towards comparative oncology is considered, emphasizing the great usefulness for the medical research to study the phylogenetic development of oncogenesis.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=130192&dopt=Abstract

Fiziol Zh SSSR Im I M Sechenova. 1976 Feb;62(2):275-82.
[The concentration of electrolytes and water in the walls of the frog gall bladder following absorption of an isotonic fluid]

[Article in Russian]

Iaremenko MS, Butusova IA.

In experiments on isolated gall-bladders (GB) of frogs, the transport rate of NaCl-isotonic solution by GB epithelial cells was inversely proportionate to the sodium content in them. Under oxytocin action (20 mE/ml) the absorption was inhibited and simultaneously the intracellular sodium content increased. Replacement of NaCl by sucrose solution caused a complete inhibition of absorption for 5--15 min. During this time, tissue cells excreted a considerable amount of their sodium, in result of which the intracellular amount of sodium decrease by 2.0-2.5 times. Oxytocin caused the inhibition of both the sodium transport from cells and the Na-K--ATPase activity. Obviously, the inhibitory action of oxytocin upon sodium transport from GB cells is due to a decrease of the transport activity of Na, K--ATPase under the action of hormone.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=130268&dopt=Abstract

Horm Res. 1975;6(3):138-44.
Testicular lesions of streptozotocin diabetic rats.

Oksanen A.

Diabetes was induced in adult male albino rats by a single intravenous injection of streptozotocin (75 mg/kg body weight). The diabetes was allowed to stabilize for at least 15 days, whereafter the testicular and seminal vesicle histology was studied at various time intervals. Reduction in testis weights and tubule diameters was significant after 2 weeks of diabetes. The changes in seminiferous tubules ranged from premature sloughing of epithelium to total cessation of spermatogenesis. The testicular histology of diabetic animals frequently greatly simulated the situation described following hypophysectomy. By subjective visual assessment the number of Leydig cells was found to be normal or reduced in all of the diabetic animals. Diabetes was also demonstrated to induce seminal vesicle atrophy, which did not show any correlation with the degree of testicular lesions. The possible etiology of testicular damage in diabetic animals is discussed.

PIP: The effects of streptozotocin-induced diabetes on the testes of adult albino rats were examined. Diabetes was induced in 118 rats by a single iv injection of streptozotocin, 75 mg/kg body weight; control animals were injected with citrate buffer. After15 days, testes were removed at varying intervals and prepared for histologic study. A mean cross-section of tubule diameters was calculated. After 30 days of diabetes, seminal vesicles were removed and similarly processed. Reduction in testes weight continued throughout the experiment (p less than .001). Seminal vesicle atrophy was complete after 30 days and the histological appearance showed changes similar to those of castrated rats. Reduction in tubule diameter was significant after 2 weeks (p less than .001). Otherwise, seminiferous tubules were histologically normal at 15 days after induction of diabetes. At 30 days 4 of 10 rats showed complete spermatogenesis. In the other 6 animals, extensive shrinkage of seminiferous tubules, massive sloughing of primary spermatocytes, and premature spermatids were observed. Peritubular connective tissue appeared intact. Atrophic changes increased with longer time periods so that at 130 days following the injections, all 7 animals studied showed extensive spermatic damage. The degree of damage was not correlated with loss of body weight or blood glucose level. Leydig cells were normal or reduced. The possible cause of diabetic testicular lesions may be hyposecretion of gonadotropic hormones or blockade of gonadotropin action.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=130334&dopt=Abstract

Probl Endokrinol (Mosk). 1975 Nov-Dec;21(6):45-9.
[Serum thyroxine-binding protein for determining the functional state of the thyroid gland in pregnant women with endemic goiter]

[Article in Russian]

Korol'kova OA, Cheremukhin VI.

A determination was made of the hormone-forming capacity of the thyroid gland in pregnent women under conditions of goiter endemic at various periods of pregnancy by trimesters (123-in healthy pregnant women, 206-with euthyroid goiter of the I degree, 271-or II degree, 90-of the II degree, and 4-of the IV degree). A method of zonal electrophoresis in the medinal-veronal buffer was applied. Thyrofixin with I131 isotope (made in the USSR) was used. With increase of the periods of pregnancy and the degree of euthyroid hyperplasia of the thyroid gland and goiter the thyroid gland function became elevated irrespective of age.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=130630&dopt=Abstract

Loss of hair changes the appearance of a person, and the identity of the person in social context to a certain extent. Hair growth is a complex biological process, which has not yet been completely understood. A multitude of therapeutic measures, including drugs, surgery, and suppelements have been made available, and used. However, due to the diversity of the problems underlying hair loss, there is no single solution for all hair loss cases. Most of chemical drugs and hair transplantation surgeries are not free from varying degrees of undesirable side effects on health.

Hair Million is an alternative solution to hair loss problems. Albeit only anecdotally, it has demonstrated efficacy in the improvement for age-related hair thinning and hair loss for a significant fraction of people who take it as recommended. We do not know the mechanisms of action as to how Hair Million works to help stop hair loss, and promote hair growth. We only know by anecdotal observations. There has been no clinical trials nor placebo controlled statistical analysis.

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