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Pathogen research abs 1 || Pathogen research abs 2 || Pathogen research abs 3 || Pathogen research abs 4 || Pathogen research abs 5 || Hormone and endocrine research abs 1 || Hormone and endocrine research abs 2 || Hormone and endocrine research abs 3 || Hormone and endocrine research abs 4 || Hormone and endocrine research abs 5

Med Clin (Barc). 2002 Jun 8;119(1):1-3.
[Usefulness of cytosolic tissue-type plasminogen activator levels in lung adenocarcinomas]

[Article in Spanish]

Ruibal A, Nunez MI, Del Rio MC, Rodriguez J, Alvarez De Linera JF.

Laboratorio de Biologia Tumoral Fundacion Jimenez Diaz, Departamento de Medicina Nuclear, Fundacion Jimenez Diaz, Madrid, Spain.

BACKGROUND: We aimed at studying the behavior of cytosol tissue-type plasminogen activator (t-PA) levels in lung adenocarcinomas and their correlation with other clinical and biological parameters. MATERIAL AND METHOD: t-PA cytosol levels were determined using EIA (Boehringer Mannheim. Germany) in 59 samples of lung adenocarcinoma and in 16 samples of normal lung tissue from the same patients. Cathepsin D, CA125, pS2, hyaluronic acid (HA), free beta subunit of chorionic gonadotropin hormone and neuron specific enolase (NSE) cytosol concentrations were determined. We also determined the concentrations of HA, erbB2 oncoprotein, epidermal growth factor receptor (EGFR), CD44s, CD44v5 and CD44v6 in cell surfaces. The following parameters were considered: clinical stage, ploidy, cellular S-phase fraction and histological grade. RESULTS: In adenocarcinomas, t-PA cytosol levels ranged from 0.1 to 14.6 ng/mg prot. (median, 1.4). These levels were lower than those in normal lung tissue (r, 0.1-18.6; median, 2.95 ng/mg prot.) but did not reach statistical significance. On the other hand, t-PA concentrations decreased as the clinical stage increased and were higher in stage I than stage II-III (p = 0.080) and stage III (p = 0.0622). No significant differences of t-PA levels were observed when the histological grade, ploidy and S-phase were considered. Adenocarcinomas with high t-PA values (> 3.7 ng/mg prot., representing the 75th percentile of the whole group), had lower CA125 (p = 0.015) and erbB2 oncoprotein (p = 0.087) concentrations. CONCLUSIONS: These results suggest that cytosol t-PA levels are negatively correlated with tumor size in lung adenocarcinomas. However, the usefulness of t-PA as a prognostic factor needs to be clarified in further studies.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12061997&dopt=Abstract

Dev Cell. 2002 Jun;2(6):721-31.
Redundant pathways for negative feedback regulation of bile acid production.

Wang L, Lee YK, Bundman D, Han Y, Thevananther S, Kim CS, Chua SS, Wei P, Heyman RA, Karin M, Moore DD.

Department of Molecular and Cellular Biology, Houston, TX 77030, USA.

The orphan nuclear hormone receptor SHP has been proposed to have a key role in the negative feedback regulation of bile acid production. Consistent with this, mice lacking the SHP gene exhibit mild defects in bile acid homeostasis and fail to repress cholesterol 7-alpha-hydroxylase expression in response to a specific agonist for the bile acid receptor FXR. However, this repression is retained in SHP null mice fed bile acids, demonstrating the existence of compensatory repression pathways of bile acid signaling. We provide evidence for two such pathways, based on activation of the xenobiotic receptor PXR or the c-Jun N-terminal kinase JNK. We conclude that redundant mechanisms regulate this critical aspect of cholesterol homeostasis.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12062085&dopt=Abstract

Aquat Toxicol. 2002 Jul;58(1-2):113-29.
Effects of 4-nonylphenol on gene expression of pituitary hormones in juvenile Atlantic salmon (Salmo salar).

Yadetie F, Male R.

Department of Molecular Biology, University of Bergen, HIB PO Box 7800, N-5020 Bergen, Norway.

Alkylphenols such as 4-nonylphenol (NP) are one of the wide variety of environmental chemicals reported to have estrogenic effects in both in vitro and in vivo studies. Induction of eggshell zona radiata proteins (Zrp) and vitellogenin (Vtg) mRNA and protein synthesis in the liver are widely used biomarkers for xenoestrogen exposure in fish. However, little work has been done to characterize the molecular effects of xenoestrogens on other potential target organs such as the pituitary. To evaluate pituitary effects and develop new potential biomarkers for xenoestrogens, the influences of NP and 17beta-estradiol (E2) on the mRNA levels of pituitary gonadotropic hormone (GTH) beta subunits [leutinizing hormone beta (LH beta or GTH II beta) and follicle stimulating hormone beta (FSH beta or GTH I beta)], prolactin (PRL), growth hormone (GH) and the pituitary specific transcription factor (Pit-1) were investigated in individual male and female juvenile Atlantic salmon (Salmo salar), 3 days after a single intraperitoneal (i.p.) injection. In one experiment, fish were injected with NP (125 mg/kg body weight (BW)) or E2 (5 mg/kg BW) and a semi-quantitative reverse transcriptase-polymerase chain reaction (RT-PCR) method was used to analyze LH beta and FSH beta mRNA levels. In the second experiment, fish were injected with three doses of NP (10, 50, 125 mg/kg BW) or a single dose of E2 (5 mg/kg BW) and Northern blot analysis was used to quantify LH beta, FSH beta, PRL, GH and Pit-1 mRNAs. Both NP (50 and 125 mg/kg BW) and E2 significantly induced LH beta mRNA levels (P<0.01), but only in females. The highest dose of NP (125 mg/kg BW) significantly induced Pit-1 mRNA in males (P<0.01). NP did not have significant effects on any of the other pituitary transcripts. NP induced LH beta mRNA synthesis in females by up to 6-fold and the changes appeared to correlate with the increases in hepatic Vtg and Zrp mRNA levels. The results show that LH beta mRNA assay in female juvenile salmonids may be used as a marker for pituitary effects of xenoestrogens. The data also suggest that NP may have the potential to perturb the regulation of LH beta gene expression by mimicking E2.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12062158&dopt=Abstract

Jpn J Ophthalmol. 2002 Mar-Apr;46(2):226-9.
Modulation of insulin-like growth factor-I production of cultured retinal vascular endothelial cells by oxygen, glucose and growth hormone.

Eter N, Sahm M, Klingmuller D, Spitznas M.

Department of Ophthalmology, Bonn University Medical Center, Bonn, Germany.

PURPOSE: To evaluate the interaction of oxygen, glucose, and growth hormone (GH) on insulin-like growth factor-I (IGF-I) production of cultured bovine retinal vascular endothelial cells. METHODS: Confluent cultures of bovine retinal vascular endothelial cells were incubated under 3% or 20% oxygen in Dulbecco's modified Eagle's medium (containing 1 g/L or 4.5 g/L glucose), with or without the addition of 10 ng/mL GH. After incubation times of 0, 24, 48, and 72 hours, IGF-I was measured in the supernatant, and cells were counted. RESULTS: Highest levels of IGF-I were reached after 72 hours with 4.5 g/L glucose and 10 ng/mL GH under 3% O(2). All IGF-I levels found in 3% O(2) samples were significantly higher than those found in 20% O(2) samples. No statistical significance was found regarding glucose concentration or GH supplement. CONCLUSIONS: Oxygen turned out to be the sole modulating factor for IGF-I production of cultured retinal vascular endothelial cells.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12062233&dopt=Abstract

Physiol Behav. 2002 Apr 1;75(4):455-64.
Glucocorticoid feedback increases the sensitivity of the limbic system to stress.

Cook CJ.

Biomedical and Bioengineering Sector, Hort Research, Private Bag 3123, Hamilton, New Zealand. ccooortresearch.co.nz

In the hypothalamus, corticotropin-releasing hormone (CRH) has a well-described role initiating the hypothalamic-pituitary-adrenal (HPA) axis response to stress. Cortisol, released from the adrenal gland, exerts negative feedback on this axis. The role of extrahypothalamic CRH in stress responses is less well known. The purpose of this study was to measure the response of CRH in the amygdala to acute and repeated stress and to examine if cortisol had any effect on this response. Immunosensor-based microdialysis probes were used to measure CRH and cortisol in the amygdala and cortisol systemically in sheep exposed to a predator stress (a dog). Upon presentation of a dog, CRH increased in the amygdala of the sheep and then fell off. Cortisol levels rose both systemically and in the amygdala, and as they peaked, a second CRH response was observed. Repeated stress changed this response, with the magnitude of the first CRH peak decreasing while the second peak increased. Repeated stress also produced an exaggeration in both of the CRH peaks to presentation of a subsequent novel stress (a forelimb electric shock). Animals that had an escape route from the repeated dog stress did not show this exaggeration when faced subsequently with the novel stress. Administration of mifepristone, a glucocorticoid receptor antagonist, prior to the delivery of the repeat stress prevented subsequent changes in the CRH response. The data suggest that the amygdala shows a CRH response to presentation of a stressor acutely and repeatedly and that repeated stress can alter subsequent amygdala responsiveness to the same or a different stressor. This alteration appears dependent on circulatory glucocorticoids.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12062310&dopt=Abstract

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