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J Cardiovasc Surg (Torino). 2002 Aug;43(4):559-61.
Severe hypoglycaemia associated with a giant solitary fibrous tumor of the pleura.

Filosso PL, Oliaro A, Rena O, Papalia E, Ruffini E, Mancuso M.

Department of Thoracic Surgery, University of Torino, San Giovanni Battista Hospital, Italy. pierluigifilossiscalinet.it

Solitary fibrous tumors (SFT) of the pleura are a rare neoplasm, with benign biological behaviour. Recurrences are rare, and no distant metastases are described in the literature. SFT can secrete hormone-like substances, responsible for paraneoplastic syndromes. The authors describe a case of severe hypoglycaemia due to insulin-like growth factor II (IGF-2)'s secretion by a giant SFT of the pleura. Hypoglycaemia was controlled by the resection of the tumor. Diagnosis and surgical management of these neoplasms are also discussed.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12124574&dopt=Abstract

Bull Exp Biol Med. 2002 Apr;133(4):339-41.
Development of insulin resistance in experimental animals during long-term glucocorticoid treatment.

Selyatitskaya VG, Kuz'minova OI, Odintsov SV.

Laboratory of Endocrinology, Research Center for Clinical and Experimental Medicine, Siberian Division of the Russian Academy of Medical Sciences, Novosibirsk. cceyber.ma.nsc.ru

Long-term treatment with glucocorticoids led to the development of insulin resistance in experimental animals, which was confirmed by a progressive increase in blood insulin level and decrease in the glucose/insulin index. The comparative study showed that hydrocortisone produced more pronounced and rapid changes than dexamethasone. However, we found no differences in the glucose/insulin index by the 23rd day of treatment with these hormones.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12124639&dopt=Abstract

Prenat Diagn. 2002 Jul;22(7):553-7.
Shortened fetal long bones: a possible in utero manifestation of placental function.

Zalel Y, Lehavi O, Schiff E, Shalmon B, Cohen S, Schulman A, Achiron R.

Department of Obstetrics and Gynecology, The Chaim Sheba Medical Center, Sackler School of Medicine, Tel-Aviv University, Tel-Hashomer, Israel. zalel_etvision.net.il

Shortened fetal long bones (SFLB) are usually indicative of a skeletal dysplasia. Our aim in this observational retrospective study was to describe a new association between SFLB, small for gestational age (SGA) fetuses and placental abnormalities, and to suggest an aetiologic explanation. During the last decade we have evaluated nine cases in which SFLB (more than 2SD below the mean) was associated with SGA, abnormal maternal serum placental hormones and abnormal placental sonography. Six cases had significantly increased second trimester maternal serum beta hCG and four developed toxaemia of pregnancy or had chronic hypertension. On histology, mature placentas with vascular abnormalities, including chorangiosis, large infarcts and slightly increased syncytial knots were noted. The combination of SFLB, SGA fetuses and placental abnormalities (sonographic, as well as histological) suggested a possible common pathway in the aetiology of this association. 2002 John Wiley & Sons, Ltd.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12124686&dopt=Abstract

J Neurobiol. 2002 Aug;52(2):99-116.
Remodeling of an identified motoneuron during metamorphosis: central and peripheral actions of ecdysteroids during regression of dendrites and motor terminals.

Knittel LM, Kent KS.

Department of Biological Structure and Function, School of Dentistry, Oregon Health & Science University, 611 S.W. Campus Drive, Portland, Oregon 97201, USA.

During metamorphosis of the moth Manduca sexta, an identified leg motoneuron, the femoral depressor motoneuron (FeDe MN), undergoes reorganization of its central and peripheral processes. This remodeling is under the control of two insect hormones: the ecdysteroids and juvenile hormone (JH). Here, we asked whether peripheral or central actions of the ecdysteroids influenced specific regressive aspects of MN remodeling. We used stable hormonal mimics to manipulate the hormonal environment of either the FeDe muscle or the FeDe MN soma. Our results demonstrate that motor-terminal retraction and dendritic regression can be experimentally uncoupled, indicating that central actions of ecdysteroids trigger dendritic regression whereas peripheral actions trigger terminal retraction. Our results further demonstrate that discrete aspects of motor-terminal retraction can also be experimentally uncoupled, suggesting that they also are regulated differently. 2002 Wiley Periodicals, Inc.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12124749&dopt=Abstract

J Comp Neurol. 2002 Aug 19;450(2):153-66.
Morphometric analysis of dendritic remodeling in an identified motoneuron during postembryonic development.

Libersat F, Duch C.

Zlotowski Center for Neuroscience and Department of Life Sciences, Ben Gurion University of the Negev, Beer Sheva 84105, Israel. libersagumail.bgu.ac.il

A detailed quantitative description of modifications in neuronal architecture is an important prerequisite to investigate the signals underlying behaviorally relevant changes in neuronal shape. Extensive morphological remodeling of neurons occurs during the metamorphosis of holometabolous insects, such as Manduca sexta, in which new adult behaviors develop postembryonically. In this study, a morphometric analysis of the structural changes of an identified Manduca motoneuron, MN5, was conducted by sampling its metric parameters at different developmental stages. The remodeling of MN5 is divided into three main phases. The regression of most larval dendrites (1) is followed by the formation of dendritic growth-cones (2), and subsequently, adult dendrite formation (3). In contrast, the cell body and link segment surface increase during dendritic regression and regrowth, indicating that different cell compartments receive different signals, or respond differently to the same signal. During dendritic growth-cone formation, the growth of the cell body and the link segment are arrested. Sholl and branch frequency analysis suggest two different modes of dendritic growth. During a first growth-cone-dependent phase, new branch formation occurs at all dendrites. The maximum path length of the major dendritic tree changes little, whereas branch order increases from 20 to 45. Changes in total dendritic length are correlated with strong changes in the number of nodes but with minor changes in the average dendritic segment length, indicating a mode of growth similar to that induced by steroid hormone application to cultured motoneurons. The second phase is growth-cone-independent, and branching is limited to high order dendrites. 2002 Wiley-Liss, Inc.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12124760&dopt=Abstract

The average human scalp is covered by approximatey 100,000 hair follicles. Each hair undergoes hair cycle and normally 50-100 hairs randomly fall out a day, which is unnoticeable because lost hair is replaced by as many new hairs springing up daily. Hair loss results from the fall out of hair from the hair follicle. Alopecia or excessive, premature hair loss is the condition caused by many factors. Loss of hair itself does not pose critical health problems because biological role of human hair is relatively marginal. Hair on our scalp protects the head from mechanical shock, heat loss, and exposure to UV-light. The eyelashes and eyebrowes protect the eyes, and hair in the ear canal or the nasal passages help filter out particles and pathogens, thus protecting our internal organs. However, hair does play important social role: it is one of the major determinants of our appearance and identity in daily life. Fullness of hair also implicates or manifests physical integrity and youthfulness of the person. Losing hair could have more than just emotional impacts on individuals. The hair is a unique organ that goes through a characteristic cycle consisting of an immature phase, a growing phase called anagen, a transitional phase between the growing phase and the resting phase called catagen, and finally a resting phase called telogen in which the hair stops growing, waiting to fall out. 85-90% of hairs on our body are in anagen phase or growing phase, which lasts anywhere from two to five years. This phase is followed by a short regression phase, or catagen, which lasts 2-3 weeks. Approximately 1% of hair follicles are in catagen. Approximately 10-15% of hair follicles are in the resting phase, the telogen, which lasts about 3-5 months. Hair follicles typically goes through 10-20 asynchronous cycles during the lifetime. Persistent loss of more than 150 hairs would consist a state of hair loss, or alopecia, albeit it could be temporary.

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