DreamPharm Products:
Lutein-20||Herbs for headache, fever, and migraine ||
Milk thistle||Saw palmetto||
Triple B Super Vision||Garlic, Ginger, and Grapeseed Extract||
Ginseng and Ginkgo||Hair Million||
DHEA||Coenzyme Q10||
Sleep Aid herbal formula - natural sleep aid||Herbal Breath - herbs for bad breath problems.||
Weight loss herbal formula for menopause and pms||Ginkgo biloba||
Colon cleansing, Laxative||ViaVita, Lecithin for healthy liver
Fatty acids resources:
Fatty acids research abs 1 || Fatty acids research abs 2 || Fatty acids research abs 3 || Fatty acids research abs 4 || Fatty acids research abs 5
Klin Med (Mosk). 2002;80(10):21-4.
[Role of thyroid pathology in pathogenesis of arterial hypertension in diabetes]
[Article in Russian]
Tereshchenko IV, Trefilova ES.
190 patients with diabetes mellitus (DM) were examined: 24 patients with DM type 1 and 166 with DM type 2. Frequency of arterial hypertension (AH) in diabetics is higher than in population. AH in different DM types varies by pathogenesis. Symptomatic renal AH is typical for DM1, essential AH combining with renal one in 1/3 of cases--for DM2. Treatment of AH and DM often provokes hypothyroidism which can be iatrogenic because of continuous intake of sugar reducing and antihypertensive drugs with antithyroid and strumogenic actions. Hypothyroidism aggravates an AH course: arterial pressure becomes high and resistant to hypotensive therapy. DM makes difficulties in selection of hypotensive drugs as many of them alter metabolism and due to negative attitude of the patients to continuous intake of sugar reducing, antihypertensive drugs, thyroid hormones. Therefore, hypertensive diabetics should be prepared for treatment psychologically and receive only prolonged hypotensive drugs. In DM with AH medication of choice is ACE inhibitors as they are nephroprotective, had no negative effect on carbohydrate, fat metabolism and thyroid system.
online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12471832&dopt=Abstract
Med Parazitol (Mosk). 2002 Jan-Mar;(1):12-6.
[Biochemical characteristics of hepatic functions in different clinical forms of chronic opisthorchiasis]
[Article in Russian]
Bakshtanovskaia IV, Stepanova TF, Shonin AL, Terekhina VK, Ponomareva OV.
The main biochemical indices of hepatic functions (the activities of alanine aminotransferase, aspartate aminotransferase, gamma-glutamyl transferase, alkaline phosphatase, alpha-amylase, choline esterase and the concentrations of total bilirubin, cholesterol, and glucose) were studied in the sera of 256 patients with chronic opisthorchiasis. It was found that with diseases manifested in different clinical forms (cholangitis, cholecystitis, cholangiocholecystitis, cholangiohepatitis, cholecystitis in combination with pancreatitis), most study indices are within the normal ranges, but significantly differ from the means in a group of apparently healthy individuals. The findings suggest that such clinical forms of opisthorchiais as cholangiocholecystitis and cholangiohepatitis are characterized by manifestations of cytolysis and cholestasis, as cholecystitis is manifested by cytolysis, as cholecystitis in combination with pancreatitis, by cholestasis, and as cholangitis, by cholestasis and hepatic cell insufficiency. It is possible that further studies will provide evidence for how to correct detected disorders during pathogenetic therapy.
online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12224256&dopt=Abstract
Med Parazitol (Mosk). 2002 Jan-Mar;(1):40-3.
[A laboratory model of the parasitic system of Lyme disease]
[Article in Russian]
Naumov RL, Vasil'eva IS, Gutova VP, Ershova AS.
A laboratory model of the parasitic system of Lyme borreliosis (LB) with Ixodes persulcatus and Borrelia burgdorferi s.s. underwent 5 epizootic cycles (Cycle I transition of Borreliae by the scheme a tick-->a mouse-->a tick corresponds to the seasonal cycle). A group of 5-6 mice was inoculated with the suspension of infected nymphs by the intracutaneously route and 35-45 days later intact tick larvae or nymphs were fed on them. In the first three cycles, 40-100% of mice in a group (a total of 33 of 59) were susceptible, but 3 of 11 groups of mice were not inoculated. 20-50% of larvae and 75-92% of nymphs were susceptible on the infected mice. The mean geometric abundance of borreliae was 2.8-6.3 cells per 100 fields of vision in the nymphs infected in the larval phase and 16.1-29.5 cells in the image phase. In cycle IV, 2 of 6 groups of mice were unsusceptible to borreliae and 5 of the 17 mice from 4 groups got infected. One mouse died from Lyme borreliosis when larvae were fed on it. The nymphs remolted from underfed larvae were infected in 62.2% of cases, the mean geometric abundance in these nymphs was 5.7% per 100 fields of vision. A total of 3.2-14.7% of the ticks fed on the remaining 4 mice were infected and the abundance of boreliae was 1-2 per 100 fields of vision. In cycle V, one of 12 mice got infected, the infection rate in nymphs was 9.1%, the abundance of borreliae was 1 per 100 fields of vision. Thus, as the number of epizootic cycles increases, the proportion of infected mice, that of ticks infected on these mice on feeding, and the abundance of borreliae in the infected ticks decrease. Moreover, there was a reduction in the length of borreliae reduced from 17-18 mm to 10-11 mm and in the duration of multiplication and preservation of borreliae in the infected ticks and there was concurrently an increase in the duration of development of an infectious process in mice. It is suggested that the population of borreliae consists of 2 morphophysiological groups or more. During cultivation of the laboratory model of the parasitic system of Lyme borreliosis, a group of borrelia that are short and slightly pathogenic for mice had got many points in its favour. A five-fold procedure of artificial election of precisely defining this group set the model on the verge of disappearance. The capacity of the populations of borreliae to alter their properties rapidly and fundamentally suggests that there might be violent annual fluctuations in human morbidity.
online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12224267&dopt=Abstract
Sheng Wu Yi Xue Gong Cheng Xue Za Zhi. 2002 Jun;19(2):181-5, 211.
[Force-dependent effects of interleukin-8 gene expression in endothelial cells exposed on fluid shear stress]
[Article in Chinese]
Zhang W, Chen H, Li L, Chen Y, Yang Y.
Institute of Biomedical Engineering, West China Medical Center of Sichuan University, Chengdu 610041.
Fluid shear stress plays an important role in many physiological and pathophysiological processes of the cardiovascular system. Previous studies have identified the exposure of vascular endothelial cells to fluid mechanical forces can modulate the expression of many genes, including IL-8 gene expression, and IL-8 expression induced by fluid shear stress was time-dependent. To investigate the role of intensity of fluid shear stress on IL-8 expression in human umbilical vein endothelial cells (HUVECs), we had HUVECs exposed to shear stress 2.23, 4.20, 6.08, 8.19, 9.67, 12.15, 14.40, 16.87, and 19.29 dyne/cm2 respectively and employed quantitative reversal transcription-polymerase chain reaction (qRT-PCR) to assay the expression of IL-8 mRNA. The results show that IL-8 mRNA did not express in HUVECs untreated with fluid shear stress. The IL-8 mRNA expression by shear stress was force intensity-dependent. After HUVECs exposed to low fluid shear stress (2.23 dyne/cm2) for 1 h or 2 h, IL-8 mRNA expression increased near 68 or 52 times as that of HUVECs exposed to high fluid shear stress (19.29 dyne/cm2). The linear regression equations between IL-8 mRNA expression (log (copies), y) and shear stress (dyne/cm2, x) are: y = 7.57 - 0.11x, r = 0.97 (for 1 h); y = 7.92 - 0.10x, r = 0.96 (for 2 h). This in vitro study demonstrates the expression of IL-8 gene can be regulated by fluid shear stress. The low shear stress could induces much more expression of IL-8 mRNA, which plays probably an important role in the pathogenesis of inflammation and arteroatherosclerosis.
online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12224276&dopt=Abstract [PubMed - in process]
Am J Health Syst Pharm. 2002 Sep 1;59(17):1626-31.
Pharmacogenomics and infectious diseases: impact on drug response and applications to disease management.
Hayney MS.
School of Pharmacy, University of Wisconsin, 777 Highland Avenue, Madison, WI 53705, USA. mshayneharmacy.wisc.edu
The impact of pharmacogenomics on the prevention, diagnosis, and treatment of infectious diseases is discussed. The application of pharmacogenomics to infectious diseases requires consideration of the genomes of both the pathogen and the host. The pathogen's genome may be used for antigen identification, to identify infecting organisms, and to determine antimicrobial resistance. Diagnostic tool development and vaccine design can be aided by knowing which portions of a pathogen are important antigenic determinants. The unique genetic makeup of a pathogen can facilitate its identification as an augmentation to the traditional culture. Important genes conferring resistance to antibiotics can be detected, and this information can be used to choose appropriate antibiotic therapy. The genome of the host may reveal susceptibility genes and new drug targets that may be used in the treatment of infectious diseases. Thus far, polymorphisms in genes of the host immune system have been associated with susceptibility to infections and response to treatment. Examples of these findings will be described. Pharmacogenomics has the potential to revolutionize the prevention, diagnosis, and treatment of infectious diseases.
online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12224343&dopt=Abstract
Natural Herbal Supplement: Hair Million
Hair Loss, or alopecia is a concern
for increasing number of folks in aging society. Loss of hair is a visible problem, and affects the appearance and changes identity of a person.
The phenomenon of hair thinning and hair loss is most commonly associated with natural aging, although there are many other causes of hair loss, which include inherited or genetic conditions, illnesses, malnutrition, stress, hormonal problems, chemotherapy, and use of some drugs.
Hair growth is a sophisticated biological process, which has not yet been completely understood. A multitude of therapeutic measures, including drugs, surgery, and suppelements have been made available, and used. However, due to the heterogeneity in the underlying cause, there is no perfect cure for all hair loss cases. Most of chemical drugs and hair transplantation surgeries are not free from varying degrees of undesirable side effects on health.
Hair Million is an alternative solution to hair loss problems. Anecdotally, it shows prositive results and improvement for age-related hair thinning and hair loss for a fraction of people who take it. We do not know the mechanisms of action as to how Hair Million works to help stop hair loss, and promote hair growth. We only know by anecdotal observations. There has been no clinical trials nor placebo controlled statistical analysis on the efficacy of Hair Million on hair loss and hair growth. However, there are two merits in this hair restoration herbal formula:
Firstly, Hair Million is rather inexpensive, and secondly, it is made of well known herbs that are safe when consumed in regular quantities.
DreamPharm Online Healthy Supplements ||
Constipation relief, laxative, colon cleansing ||
Lutein ||
Progesterone Cream ||
Natural herbal formula for hair loss problems ||