DreamPharm Products:
Lutein-20||Herbs for headache, fever, and migraine ||
Milk thistle||Saw palmetto||
Triple B Super Vision||Garlic, Ginger, and Grapeseed Extract||
Ginseng and Ginkgo||Hair Million||
DHEA||Coenzyme Q10||
Sleep Aid herbal formula - natural sleep aid||Herbal Breath - herbs for bad breath problems.||
Weight loss herbal formula for menopause and pms||Ginkgo biloba||
Colon cleansing, Laxative||ViaVita, Lecithin for healthy liver
Fatty acids resources:
Fatty acids research abs 1 || Fatty acids research abs 2 || Fatty acids research abs 3 || Fatty acids research abs 4 || Fatty acids research abs 5
Rev Neurol. 2002 Aug 1;35(3):212-20.
[Molecular basis of Huntington s disease and possible pathogenic mechanisms]
[Article in Spanish]
Martin Aparicio E, Lucas Lozano JJ.
Centro de Biologia Molecular "Severo Ochoa". CSIC/UAM, Cantoblanco, Espa a.
INTRODUCTION. Huntington s disease is one of the, at least, nine neurological disorders caused by a CAG triplet expansion coding for a poly glutamine sequence in the corresponding protein. Huntington s disease affects 3 7 in 100.000 individuals in Western Europe descendent population and the symptomatology comprises motor (including chorea and rigidity), cognitive (subcortical dementia), and psychological (including irritability and depression) manifestations until death. DEVELOPMENT. Neuropathology is extremely restricted, with atrophy occurring in the striatum and, to a lesser extent, in the cerebral cortex. Microscopically, the neuropathology is characterized by neuronal loss, reactive gliosis, and intraneuronal protein aggregates. Since the initial description of this disease by George Huntington in 1872, substantial advance has been achieved in the understanding of this pathology. The pathogenic gene and mutation were identified in 1993. This allowed the generation of multiple in vitro, cellular, and animal models of Huntington s disease. These studies have originated multiple hypotheses regarding the mechanism by which huntingtin with an expanded poly glutamine tract exerts its toxicity. CONCLUSION. We try to summarize the current knowledge about this disease from the clinical manifestations to the molecular basis, in an attempt to offer a global view of this pathology.
online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12235581&dopt=Abstract [PubMed - in process]
Rev Neurol. 2002 Aug 1;35(3):269-76.
[Recent aspects of acute and chronic inflammatory polyneuropathies: Guillain Barr syndrome and chronic inflammatory demyelinating polyneuropathy]
[Article in Spanish]
Pascual Pascual SI.
Hospital Ntra Sra de Sonsoles, avila, Espa a.
INTRODUCTION. In last decade many advances have occurred in knowledge of pathogenic, in the different types of clinic expression and in the therapy of both acute and chronic polyneuritis. OBJECTIVE. To review the recent advances in the childhood expression of these disorders. DEVELOPMENT AND CONCLUSIONS. Into the broad term of Guillain Barr syndrome (GBS) several types are considered: demyelinating, motor sensory axonal and motor axonal, and the Miller Fisher syndrome (MFS). Diagnostic criteria, clinical and neurophysiological are explained. The actual modes of treatment are reviewed, especially immunoglobulins and plasmapheresis. The chronic inflammatory demyelinating polyneuropathy (CIDP) is a rare disease in childhood but it is necessary think on it when a picture of demyelinating polyneuropathy that seems the more frequent Charcot Marie Tooth type 1 polyneuropathy, because the treatment of CIDP changes radically the prognosis. Last advances of pathogenic, diagnostic criteria and treatment with corticosteroids, immunoglobulins and plasmapheresis are reviewed.
online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12235589&dopt=Abstract [PubMed - in process]
Rev Neurol. 2002 Aug 1;35(3):277-8.
[Neurological complications of non neurological disorders: from protagonism to comorbidity]
[Article in Spanish]
Garaizar C.
Hospital de Cruces, Barakaldo, Espa a.
INTRODUCTION. The development of scientific advances, constantly opening new frontiers of knowledge, leads to increasing complexity and specialization. DEVELOPMENT. The conflict between the never ending medical specialization and the opposite tendency, which is based mostly on economic grounds, prevents the acquisition of new knowledge across different disorders and diseases. Considering the brain as a complex adaptative system, it follows that in order to understand its pathogenic mechanisms and to design new therapies, the study of the interrelation of all parts concerned must be undertaken. Such approach requires the use of increasingly complex degrees of knowledge, covering different fields and disciplines. A first step might be the study of neurological complications of non neurological disorders. The former should leave its principal status in order to be studied as a consequence of the latter by disconcerted neurologists. The neurological complications of systemic cancer, congenital cardiopathy and autoinmune diseases are good examples of the need for a multidisciplinary approach to achieve a satisfactory capacity of diagnosis and treatment.
online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12235590&dopt=Abstract [PubMed - in process]
Kidney Int. 2003 Jan;63(1):209-16.
Mycophenolate mofetil prevents the development of glomerular injury in experimental diabetes.
Utimura R, Fujihara CK, Mattar AL, Malheiros DM, De Lourdes Noronha I, Zatz R.
Department of Clinical Medicine, Faculty of Medicine, University of Sao Paulo, Sao Paulo, Brazil.
BACKGROUND: Experimental and clinical evidence suggests that inflammation plays a role in the pathogenesis of diabetic nephropathy, in addition to, or in concert with, the associated hemodynamic and metabolic changes. The present study assessed the effects of chronic anti-inflammatory therapy in experimental diabetic nephropathy. METHODS: Adult male Munich-Wistar rats were made diabetic with streptozotocin after uninephrectomy, kept moderately hyperglycemic by daily injections of NPH insulin and distributed among three groups: C, non-diabetic rats; DM, rats made diabetic and treated with insulin as described earlier; and DM+MMF, diabetic rats receiving insulin and treated with mycophenolate mofetil (MMF), 10 mg/kg once daily by gavage. Renal hemodynamic studies were performed 6 to 8 weeks after induction of diabetes. Additional rats were followed during 8 months, at the end of which renal morphological studies were performed. RESULTS: After 6 to 8 weeks, diabetic rats exhibited marked glomerular hyperfiltration and hypertension. Diabetic rats developed progressive albuminuria and exhibited widespread glomerulosclerotic lesions associated with macrophage infiltration at 8 months. Treatment with MMF had no effect on blood pressure, glomerular dynamics or blood glucose levels, but did prevent albuminuria, glomerular macrophage infiltration and glomerulosclerosis. Thus, the renoprotective effect of MMF was not associated with a metabolic or renal hemodynamic effect, and must have derived from its well-known anti-inflammatory properties, which include restriction of lymphocyte and macrophage proliferation and limitation of the expression of adhesion molecules. CONCLUSIONS: These findings are consistent with the notion that inflammatory events are central to the pathogenesis of diabetic nephropathy and suggest that MMF may help prevent the progression of diabetic nephropathy.
online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12472785&dopt=Abstract [PubMed - in process]
Rocz Panstw Zakl Hig. 2002;53(2):149-56.
[Microbiologic evaluation of instant soup concentrates]
[Article in Polish]
Wojcik-Stopczynska B, Falkowski J, Jakubowska B.
Katedra Technologii Rolnej i Przechowalnictwa Akademii Rolniczej w Szczecinie 71-434 Szczecin, ul. Slowackiego 17.
The microbiological condition of instant soup powders purchased in retail network has been assessed. The study included 37 instant soups (8 types) manufactured by four Polish companies. The microbiological quality of a majority of soup powders fulfilled the requirements of the standard. No pathogenic bacteria (Salmonella, E. coli and Staphylococcus aureus) were detected, nor were there any spores of sulphitereducing anaerobic bacteria found. However, some samples of powders, mainly from one manufacturer, did have an excessive total number of bacteria (> 10(5) cfu/g) and a reduced (down to 0.01 g) level of coliform count. Aerobic bacteria occurring in powders were of vegetative and spore forms and exhibited the activity of amylo-, lipo- and proteolytic exoenzymes. The quantity of moulds did not exceed 100 cfu/g in a majority of samples. They were mainly represented by Penicillium sp., Aspergillus sp., Alternaria sp. and Cladosporium sp.
online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12235671&dopt=Abstract
Is your hair shedding prematurely? Are you losing hair gradually or all of a sudden, or just as you are aging?
Hair Million is a herbal formula to reverse your hair loss problems.
Numerous anecdotal cases, and personal testimonies indicate that this herbal formula based on Chinese herbs actually
improves the conditions of hair thinning and hair loss for a significant fraction of people taking it regularly.
The biology of hair growth is complex and a field still under exploration.
We don't know how Hair Million stops hair loss, and promotes
hair restoration, despite all the supporting anecdotal cases.
Neither scientific research nor placebo controlled clinical trials has been conducted due to the cost. Shortage of scientific
and clinical research data is not uncommon in herbal and nutritional arena. Important merits of Hair Million is that it is relatively
inexpensive compared with surgical transplantation or prescription drugs, and secondly, it is made only of traditional herbs that
promote hair growth and are widely known to be safe in regular quantities. If you are interested in clinically tested prescription
DHEA is a natural hormone, and it is produced in our body by the adrenal glands.
DHEA has been suggested to provide numerous potential benefits. DHEA (or dehydroepiandrosterone) is converted into androgens (male hormones)
or estrogens (female hormones) in the cells.
Our bodies produce decreasing amount of DHEA as we get older.
various health benefits: To deter aging,
improve sexual function/erectile dysfunction, treat cognitive decline, enhance athletic performance,
facilitate weight loss, improve strength, prevent osteoporosis, enhance immunomodulation for rheumatic conditions,
and treat depression.
DreamPharm Online Healthy Supplements ||
Constipation relief, laxative, colon cleansing ||
Lutein ||
Progesterone Cream ||
Natural herbal formula for hair loss problems ||