DreamPharm Products:
Lutein-20||Herbs for headache, fever, and migraine ||
Milk thistle||Saw palmetto||
Triple B Super Vision||Garlic, Ginger, and Grapeseed Extract||
Ginseng and Ginkgo||Hair Million||
DHEA||Coenzyme Q10||
Sleep Aid herbal formula - natural sleep aid||Herbal Breath - herbs for bad breath problems.||
Weight loss herbal formula for menopause and pms||Ginkgo biloba||
Colon cleansing, Laxative||ViaVita, Lecithin for healthy liver
Fatty acids resources:
Fatty acids research abs 1 || Fatty acids research abs 2 || Fatty acids research abs 3 || Fatty acids research abs 4 || Fatty acids research abs 5
Rev Esp Enferm Dig. 2003 Mar;95(3):233-6, 229-32.
A continuous spectrum of neutrophilic dermatoses in Crohn's disease.
[Article in English, Spanish]
Mendoza JL, Garcia-Paredes J, Pena AS, Cruz-Santamaria DM, Iglesias C, Diaz Rubio M.
Servicio de Aparato Digestivo. Hospital Clinico San Carlos. Madrid. Spain. jmendozaeditex.es
The inflammatory bowel disease is accompanied by cutaneous manifestations in approximately 10% of the cases. Neutrophilic dermatoses are located on the dermis and/or epidermis and are characterised on histological examination by the presence of an infiltrate that consists largely of neutrophils. The prototype of neutrophilic dematoses is Sweetacute;s syndrome; which is rarely associated with Crohns disease. CASE REPORT: A 63 year old woman was admitted to hospital with pyrexia, abdominal pain, episcleritis and skin lesions. She presented erythematous lesions on trunk, legs and arms, with tendency towards formation of plaques, nodules and vesicular pustular lesions. Both the colonoscopy and colonic biopsies confirmed the diagnosis of colonic Crohns disease. Cutaneous biopsies re-vealed an infiltrate consisting mainly of neutrophils. These biopsies, together with clinical details led to the diagnosis of Sweetacute;s syndrome. A methylprednisolone treatment rapidly improved the skin lesions and clinical symptoms. The different clinical forms of neutrophilic dermatosis are an extra intestinal manifestation of Crohns disease, and are some-times found concurrently in the same patient, which would indicate a common pathogenesis with different clinical presentations (spectrum of neutrophilic dermatoses).
online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12760712&dopt=Abstract
Rev Esp Enferm Dig. 2003 Feb;95(2):135-42, 127-34.
Hepatic encephalopathy: nomenclature, pathogenesis and treatment.
[Article in English, Spanish]
Quero Guillen JC, Carmona Soria I, Garcia Montes JM, Jimenez Saenz M, Herrerias Gutierrez JM.
Servicio de Aparato Digestivo. Hospital Virgen Macarena. Sevilla. Spain. queroguilleahoo.es
Hepatic encephalopathy (HE) is a neuropsychiatric syndrome in patients with liver failure and/or a portal-systemic bypass. Since 2002 a new nomenclature of HE exists, that classifies HE in encephalopathy type A (associated with acute liver failure), type B (associated with portal-systemic bypass), and type C (associated with liver cirrhosis). HE type A is characterized by a rapid development to coma, cerebral edema, and a poor short-term prognosis. Therefore, these patients should be referred to a liver transplantation center. Standard treatment of HE consists of non absorbable disaccharides, non absorbable antibiotics, and a diet with an appropriate amount of proteins. In addition, the possibility of performing a liver transplantation should be evaluated. In patients with intractable HE other alternative treatments adjunct to standard treatment, like zinc, sodium benzoate, ornithine aspartate, branched chain amino acids, flumazenil, and bromocriptine should be considered.
online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12760720&dopt=Abstract
Zhonghua Kou Qiang Yi Xue Za Zhi. 2003 Jan;38(1):27-30.
[Cloning and polymorphism analysis of prtH gene from Porphyromonas gingivalis]
[Article in Chinese]
Zheng Y, Yang SH, Zhou W, Zhang CM, Zhang FP, Dong XP.
Beijing Institute of Dental Research, Beijing Hospital for Stomatology, Capital University of Medical Sciences, Beijing 100050, China.
OBJECTIVE: To clone the prtH gene from Porphyromonas gingivalis (P.g) ATCC 33277 and analyze the polymorphism of prtH gene from 5 strains of P.g in order to explore the relationship between P.g and periodontitis. METHODS: Using PCR, the prtH was amplified and cloned into pGEM-T vector. To illustrate the prtH polymorphism among P.g strains, the genomic DNAs were extracted and screened by PCR with three pairs of specific primers, dot blot and Southern blot hybridization using the biotin-labeled prtH sequence as probe. RESULTS: Recombinant DNA pGEM-T- prtH was verified by restriction endonuclease and sequence assay. Strain W 381 and ATCC 33277 showed the identical results in PCR and hybridization assays, whereas strain ATCC 49417 and 14-3-2 revealed individual hybridization patterns. Strain 47A-1 seemed even not to contain prtH gene. CONCLUSIONS: Different prtH gene sequences exist in different P.g strains. This polymorphism may indicate various potential virulent effects during the infection and pathogenesis. Established PCR protocol is sensitive for identification of prtH gene.
online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12760772&dopt=Abstract [PubMed - in process]
Zhonghua Yu Fang Yi Xue Za Zhi. 2003 Jan;37(1):37-40.
[Role of hepatitis B virus infection in pathogenesis of IgA nephropathy]
[Article in Chinese]
Wang NS, Wu ZL, Zhang YE, Guo MY, Liao LT.
Department of Nephrology, The Sixth People's Hospital of Shanghai, Shanghai 200233, China.
OBJECTIVE: To clarify the relationship between hepatitis B virus (HBV) infection and IgA nephropathy (IgAN). METHODS: HBV antigen (HBAg) in renal tissues of the patients with IgAN was detected by immunohistochemical technique, the carrier status and localization of HBV DNA in renal tissues were determined by Southern blot analysis and in situ hybridization. RESULTS: Serum HBsAg was detected in 18 of the 100 patients with IgAN (18%), HBAg was detected in 31 of 100 patients (31%) in their renal tissue and in 20 of 31 patients (65%) in their glomeruli, and both HBsAg and HBcAg were detected in 10 of 31 patients (32%), respectively. HBcAg was also found in tubular epithelia (45%, 14/31) and renal interstitium (6%, 2/31), respectively. Five of six cases were proved to be positive of integrated-form HBV DNA in their renal tissue by Southern blot analysis. In situ hybridization demonstrated that HBV DNA was 8/8 and 6/8 positive in their renal tubules and glomeruli of all eight specimens, localized in the nucleus of tubular epithelial cells, glomerular mesangial cells, as well as infiltrated interstitial lymphocytes. CONCLUSION: HBV infection closely related with IgAN and HBV infection might be involved in pathogenesis of IgAN.
online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12760794&dopt=Abstract
Zhonghua Bing Li Xue Za Zhi. 2003 Feb;32(1):10-3.
[Mutation of 5'noncoding region of the bcl-6 gene in diffuse large B cell lymphomas]
[Article in Chinese]
Zhou XY, Zhu WP, Zhang TM, Li XM, Jin AP, Sun MH, Zhu XZ.
Laboratory of Molecular Pathology, Cancer Hospital, Fudan University, Shanghai 200032, China.
OBJECTIVE: To observe the mutation of 5'noncoding region of bcl-6 gene in diffuse large B cell lymphoma (DLBCL) and its effect on lymphoma pathogenesis. METHODS: 38 DLBCL, 2 reactive hyperplasias, 5 follicular lymphomas and 5 T cell lymphomas were chosen for PCR direct sequence analysis using two sets of primers in 5'noncoding region of the bcl-6 gene. RESULTS: No mutation was found in the marginal region of reactive hyperplasias, T cell lymphomas, and follicular lymphomas but detected in 1/2 of the follicular center cells, and 7/38 cases of DLBCL. The incidence is less than that seen in other reports. Basepairs substitution and point insertion were the main mutation types. CONCLUSIONS: The positive rate of mutation of 5'noncoding region of bcl-6 gene in DLBCL is 18.7%, less frequent than the published data of DLBCL reported in other countries. It may, in some extent, participate in the pathogenesis and progression of DLBCL.
online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12760796&dopt=Abstract [PubMed - in process]
Prescription drugs, surgical hair transplantation, topical application of various oils or creams... Also prayer and wishing...
Hair Million is an alternative approach to hair loss problems.
Anecdotes and personal experiences testify that it works. Hair Million shows positive results and improvement for age-related
hair thinning and hair loss for a large fraction of people who take it.
How does it work? Good question. The molecular biological or clinical mechanisms of action as to how Hair Million exactly works
to help stop hair loss, and promote hair growth is completely unknown.
The only evidences for the effecacy of Hair Million on hair growth are only anedotal and based on personal experiences.
There has been no clinical trials or placebo controlled statistical analysis on the efficacy of Hair Million on hair loss and hair growth.
That's enough for many people. Also, there are two merits in the hair restoration herbal formula:
Firstly, HairMillion is comparatively inexpensive, and secondly, it is made only of herbs
that are known to be safe when consumed in regular quantities. Herbs in Hair Million are also known for cardiotonic effects, meaning
that the herbs will make your heart stronger.
DHEA is a natural hormone, and it is produced in our body by the adrenal glands.
DHEA has been suggested to provide numerous potential benefits. DHEA (or dehydroepiandrosterone) is converted into androgens (male hormones)
or estrogens (female hormones) in the cells.
DreamPharm Online Healthy Supplements ||
Lutein ||
Celexa Online || Paxil Online || Buspar Online || Tramadol Online || Flexeril Online ||
Natural herbal formula for hair loss problems ||