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Curr Infect Dis Rep. 2000 Aug;2(4):302-307.
Immunogenic and Genetic Factors in Rheumatic Fever.

Carreno-Manjarrez R, Visvanathan K, Zabriskie JB.

Laboratory of Clinical Microbiology and Immunology, The Rockefeller University, 1230 York Avenue, New York, NY 10021, USA. zabrisockvax.rockefeller.edu

Although rheumatic fever has virtually disappeared in many developed countries, it remains the number one acquired heart disease among children in developing countries. Many hypotheses advanced to explain the pathogenic mechanisms of this disease include the concept of an abnormal humoral and cellular response within the host to streptococcal antigens cross-reacting with mammalian tissues. This report examines the most recent evidence supporting this concept. It emphasizes the importance of the cellular arm of the immune response in cardiac damage, as wall as possible genetic disposition to the disease.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=11095869&dopt=Abstract [PubMed - as supplied by publisher]

Curr Infect Dis Rep. 2000 Aug;2(4):315-321.
Pathogenic Mechanisms of Enterococcal Endocarditis.

McCormick JK, Hirt H, Dunny GM, Schlievert PM.

Department of Microbiology, University of Minnesota Medical School, 420 Delaware Street, SE, Minneapolis, MN 55455, USA. patenti.med.umn.edu

Enterococci are gram-positive bacteria that are now established as major nosocomial pathogens and have become increasingly important in recent years due to the development and transmission of antibiotic resistance traits. These organisms commonly cause a variety of nosocomial infections, including surgical wound infections and urinary tract infections, as well as cardiovascular infections such as bacteremia and endocarditis. Infective endocarditis is a life-threatening microbial infection of the endothelial surface of the heart, which typically occurs on heart valve tissue. The enterococci are the third most common cause of infective endocarditis, and are becoming increasingly significant in this disease. In this review, we discuss the role of enterococci in infective endocarditis and focus on the current knowledge of enterococcal virulence mechanisms, with specific reference to this disease.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=11095871&dopt=Abstract [PubMed - as supplied by publisher]

Theor Appl Genet. 2003 Jan;106(2):287-92.
Mapping of a resistance gene effective against Karnal bunt pathogen of wheat.

Sukhwinder-Singh, Brown-Guedira GL, Grewal TS, Dhaliwal HS, Nelson JC, Singh H, Gill BS.

Department of Plant Pathology, Kansas State University, Manhattan, KS 66506, USA.

A set of 130 wheat recombinant inbred lines (RILs) developed from a cross between parents susceptible (WL711) and resistant (HD29) to Karnal bunt (caused by Tilletia indica), were screened for 3 years with the pathogen populations prevalent in northern India. When 90 simple sequence repeats (SSRs) and 81 amplified fragment length polymorphism (AFLP) loci were mapped on the RILs, markers on chromosomes 2A, 4B and 7B accounted collectively for about one-third of the variation in the disease reaction. The genomic region of largest effect, identified on the long arm of chromosome 4B, reduced Karnal bunt disease by half in three different experiments and accounted for up to 25% of the phenotypic variation for KB reaction. A closely linked SSR marker, GWM538, may be useful in marker-assisted selection for Karnal bunt resistance in wheat.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12596729&dopt=Abstract

Curr Infect Dis Rep. 2000 Aug;2(4):332-336.
New Advances in the Pathogenesis and Pathophysiology of Bacterial Meningitis.

Nathan BR, Scheld WM.

Department of Neurology, University of Virginia School of Medicine, PO Box 394, Charlottesville, VA 22908, USA. brn3irginia.edu

Acute bacterial meningitis continues to be a significant health concern, with a fatality rate of more than 30% in some studies. Although the face of bacterial meningitis has changed substantially over the past 15 years, this disease still causes significant mortality (particularly in underdeveloped countries) and neurological sequelae. Our understanding of the pathophysiology of bacterial meningitis continues to develop. Our understanding of the mechanisms of neuronal injury now includes the concept that many of the pathological changes are only secondary to the infection and that the human immune system contributes to the majority of the neuronal death. A complicated series of interactions among immune, vascular and central nervous system cells, cytokines and chemokines, matrix metalloproteinases and free radical molecules are ultimately responsible for many bacterial meningitis changes. We hope that a complete understanding of these processes will ultimately lead to better diagnostic techniques and improved treatments.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=11095874&dopt=Abstract [PubMed - as supplied by publisher]

Curr Infect Dis Rep. 2000 Aug;2(4):337-344.
Bacterial Meningitis: Principles and Practical Aspects of Therapy.

Aronin SI.

Department of Internal Medicine, Section of Infectious Diseases, Waterbury Hospital Health Center, 64 Robbins Street, Waterbury, CT 06721, USA. saronitbyhosp.chime.org

Acute bacterial meningitis continues to be associated with significant morbidity and mortality. Recent research efforts to improve outcome for such patients have focused on antibiotic-cerebrospinal fluid interactions, factors influencing antibiotic selection, indications for cranial imaging, the role of anti-inflammatory agents, and the impact of antibiotic timing on outcome. In this article, I use an illustrative case to review timely data on these topics and provide guidelines for the empiric and pathogen-directed treatment of patients with acute bacterial meningitis.

online pharmacy ref. source: www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=11095875&dopt=Abstract [PubMed - as supplied by publisher]

Loss of hair changes the appearance of a person, and the identity of the person in social context to a certain extent. Hair growth is a complex biological process, which has not yet been completely understood. A multitude of therapeutic measures, including drugs, surgery, and suppelements have been made available, and used. However, due to the diversity of the problems underlying hair loss, there is no single solution for all hair loss cases. Most of chemical drugs and hair transplantation surgeries are not free from varying degrees of undesirable side effects on health.

Hair Million is an alternative solution to hair loss problems. Albeit only anecdotally, it has demonstrated efficacy in the improvement for age-related hair thinning and hair loss for a significant fraction of people who take it as recommended. We do not know the mechanisms of action as to how Hair Million works to help stop hair loss, and promote hair growth. We only know by anecdotal observations. There has been no clinical trials nor placebo controlled statistical analysis.

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